Calciprotein Particles Cause Endothelial Dysfunction under Flow

Calciprotein particles (CPPs), which increasingly arise in the circulation during the disorders of mineral homeostasis, represent a double-edged sword protecting the human organism from extraskeletal calcification but potentially causing endothelial dysfunction. Existing models, however, failed to d...

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Veröffentlicht in:	International journal of molecular sciences 2020-11, Vol.21 (22), p.8802
Hauptverfasser:	Shishkova, Daria, Markova, Victoria, Sinitsky, Maxim, Tsepokina, Anna, Velikanova, Elena, Bogdanov, Leo, Glushkova, Tatiana, Kutikhin, Anton
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Blood circulation Calcification Calcifying Nanoparticles - adverse effects Calcium - chemistry Cardiovascular diseases Cell adhesion Cell adhesion & migration Cell adhesion molecules Cell culture Cells, Cultured Coronary vessels Cytokines Endothelial cells Endothelial Cells - pathology Gene expression Homeostasis Humans Inflammation Intercellular adhesion molecule 1 Intravenous administration Investigations KLF4 protein Krueppel-like factor Leukocytes (mononuclear) Male Mechanotransduction Mechanotransduction, Cellular Mesenchyme Peripheral blood mononuclear cells Rats Rats, Wistar Risk analysis Risk factors Smooth muscle Snail protein Stress, Mechanical Transcription factors Vascular Diseases - metabolism Veins & arteries Western blotting Yes-associated protein
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