Presynaptic GABA(B) receptors decrease neurotransmitter release in vestibular nuclei neurons during vestibular compensation

Unilateral damage to the peripheral vestibular receptors precipitates a debilitating syndrome of oculomotor and balance deficits at rest, which extensively normalize during the first week after the lesion due to vestibular compensation. In vivo studies suggest that GABA(B) receptor activation facili...

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Veröffentlicht in:	Neuroscience 2012-10, Vol.223, p.333
Hauptverfasser:	Shao, M, Reddaway, R, Hirsch, J C, Peusner, K D
Format:	Artikel
Sprache:	eng
Schlagworte:	6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology Animals Animals, Newborn Baclofen - pharmacology Chick Embryo Excitatory Amino Acid Antagonists - pharmacology GABA Agents - pharmacology In Vitro Techniques Lysine - analogs & derivatives Microscopy, Confocal Microtubule-Associated Proteins - metabolism Neurons - cytology Neurons - drug effects Neurons - metabolism Neurotransmitter Agents - metabolism Patch-Clamp Techniques Presynaptic Terminals - drug effects Presynaptic Terminals - metabolism Receptors, GABA-B - metabolism Sodium Channel Blockers - pharmacology Statistics, Nonparametric Synaptic Potentials - drug effects Synaptotagmin I - metabolism Synaptotagmin II - metabolism Tetrodotoxin - pharmacology Time Factors Valine - analogs & derivatives Valine - pharmacology Vestibular Diseases - pathology Vestibular Nuclei - pathology
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