Diabetes induces and calcium channel blockers prevent cardiac expression of proapoptotic thioredoxin-interacting protein

Department of Medicine, University of Wisconsin and William F. Middleton Veterans Administration Hospital, Madison, Wisconsin Submitted 24 November 2008 ; accepted in final form 17 February 2009 Cardiomyocyte apoptosis is a critical process in the pathogenesis of ischemic and diabetic cardiomyopathy...

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Veröffentlicht in:	American journal of physiology: endocrinology and metabolism 2009-05, Vol.296 (5), p.E1133-E1139
Hauptverfasser:	Chen, Junqin, Cha-Molstad, Hyunjoo, Szabo, Anna, Shalev, Anath
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apoptosis Apoptosis - drug effects Apoptosis - physiology Biochemistry Calcium Calcium Channel Blockers - pharmacology Carrier Proteins - biosynthesis Caspase 3 - metabolism Cell Line Cell Survival - physiology Diabetes Diabetes Mellitus, Experimental - metabolism Diabetes Mellitus, Type 2 - metabolism Glucose - metabolism Glucose - pharmacology Heart Immunoblotting In Situ Nick-End Labeling Male Mice Mice, Inbred C57BL Mice, Obese Myocytes, Cardiac - metabolism Proteins Rats Rodents Thioredoxins - biosynthesis Verapamil - pharmacology
Online-Zugang:	Volltext
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