Ischemic defects in the electron transport chain increase the production of reactive oxygen species from isolated rat heart mitochondria

1 Divisions of Cardiology and 2 Clinical Pharmacology, Department of Medicine, 3 Department of Pharmacology, School of Medicine, Case Western Reserve University, and 4 Medical Service, Louis Stokes Department of Veterans Affairs Medical Center, Cleveland, Ohio Submitted 22 May 2007 ; accepted in fin...

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Veröffentlicht in:	American Journal of Physiology: Cell Physiology 2008-02, Vol.294 (2), p.C460-C466
Hauptverfasser:	Chen, Qun, Moghaddas, Shadi, Hoppel, Charles L, Lesnefsky, Edward J
Format:	Artikel
Sprache:	eng
Schlagworte:	Anemia Animals Cardiology Cardiovascular disease Cells Electron transfer Electron Transport - drug effects Electron Transport Chain Complex Proteins - metabolism Electron Transport Complex I - drug effects Electron Transport Complex I - metabolism Electron Transport Complex III - drug effects Electron Transport Complex III - metabolism Glutamic Acid - metabolism Heart Hydrogen Peroxide - metabolism Ischemic Preconditioning Male Mitochondria, Heart - metabolism Myocardial Ischemia - metabolism Myocardial Ischemia - physiopathology Myocardium - metabolism Oxidative Stress - physiology Oxygen Protein Synthesis Inhibitors - pharmacology Rats Rats, Inbred F344 Reactive Oxygen Species - metabolism Reperfusion Injury - metabolism Reperfusion Injury - physiopathology Rodents Rotenone - pharmacology Succinic Acid - metabolism Uncoupling Agents - pharmacology
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