Deficiency of M2 muscarinic acetylcholine receptors increases susceptibility of ventricular function to chronic adrenergic stress

1 Division of Basic Biomedical Sciences, Sanford School of Medicine, University of South Dakota, Vermillion, South Dakota; and 2 Molecular Signaling Section, Laboratory of Bioorganic Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Bethesda,...

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Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 2008-02, Vol.294 (2), p.H810-H820
Hauptverfasser:	LaCroix, Carly, Freeling, Jessica, Giles, Alese, Wess, Jurgen, Li, Yi-Fan
Format:	Artikel
Sprache:	eng
Schlagworte:	Adrenergic beta-Agonists - pharmacology Animals Blood Pressure - drug effects Blotting, Western Cells, Cultured Chronic Disease Heart - physiopathology Heart Rate - physiology Heart Ventricles Isoproterenol - pharmacology Matrix Metalloproteinases - metabolism Mice Mice, Knockout Mitogen-Activated Protein Kinases - metabolism Muscarinic Agonists - pharmacology Myocytes, Cardiac - drug effects Myocytes, Cardiac - physiology Phenylephrine - pharmacology Phosphorylation Rats Rats, Sprague-Dawley Receptor, Muscarinic M2 - genetics Receptor, Muscarinic M2 - physiology Reverse Transcriptase Polymerase Chain Reaction RNA - biosynthesis RNA - genetics Stress, Physiological - physiopathology Sympathetic Nervous System - physiopathology Tissue Inhibitor of Metalloproteinases - biosynthesis Tissue Inhibitor of Metalloproteinases - genetics Vasoconstrictor Agents - pharmacology Ventricular Function, Left - physiology
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