Point:Counterpoint "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction"
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Veröffentlicht in: | Journal of applied physiology (1985) 2007-05, Vol.102 (5), p.2078 |
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container_issue | 5 |
container_start_page | 2078 |
container_title | Journal of applied physiology (1985) |
container_volume | 102 |
creator | Packer, C Subah Pelaez, Nancy J |
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identifier | ISSN: 8750-7587 |
ispartof | Journal of applied physiology (1985), 2007-05, Vol.102 (5), p.2078 |
issn | 8750-7587 |
language | eng |
recordid | cdi_pubmed_primary_17549821 |
source | MEDLINE; American Physiological Society; Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals; Alma/SFX Local Collection |
subjects | Animals Calcium - metabolism Calcium Signaling Endothelin-1 - metabolism Endothelins - metabolism Endothelium, Vascular - metabolism Humans Hypoxia - metabolism Hypoxia - physiopathology Intracellular Signaling Peptides and Proteins - metabolism Muscle, Smooth, Vascular - metabolism Protein-Serine-Threonine Kinases - metabolism Pulmonary Artery - enzymology Pulmonary Artery - metabolism rho-Associated Kinases rhoA GTP-Binding Protein - metabolism Vasoconstriction |
title | Point:Counterpoint "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction" |
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