Point:Counterpoint "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction"

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Veröffentlicht in:Journal of applied physiology (1985) 2007-05, Vol.102 (5), p.2078
Hauptverfasser: Packer, C Subah, Pelaez, Nancy J
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creator Packer, C Subah
Pelaez, Nancy J
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identifier ISSN: 8750-7587
ispartof Journal of applied physiology (1985), 2007-05, Vol.102 (5), p.2078
issn 8750-7587
language eng
recordid cdi_pubmed_primary_17549821
source MEDLINE; American Physiological Society; Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals; Alma/SFX Local Collection
subjects Animals
Calcium - metabolism
Calcium Signaling
Endothelin-1 - metabolism
Endothelins - metabolism
Endothelium, Vascular - metabolism
Humans
Hypoxia - metabolism
Hypoxia - physiopathology
Intracellular Signaling Peptides and Proteins - metabolism
Muscle, Smooth, Vascular - metabolism
Protein-Serine-Threonine Kinases - metabolism
Pulmonary Artery - enzymology
Pulmonary Artery - metabolism
rho-Associated Kinases
rhoA GTP-Binding Protein - metabolism
Vasoconstriction
title Point:Counterpoint "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction"
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