Autoregulation of CCL26 synthesis and secretion in A549 cells: a possible mechanism by which alveolar epithelial cells modulate airway inflammation

Florida A & M University, College of Pharmacy and Pharmaceutical Sciences, Tallahassee, Florida Submitted 19 January 2005 ; accepted in final form 25 April 2005 Eotaxins (CCL11, CCL24, CCL26) originating from airway epithelial cells and leukocytes have been detected in bronchoalveolar lavage of...

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Veröffentlicht in:	American journal of physiology. Lung cellular and molecular physiology 2005-09, Vol.289 (3), p.L478-L488
Hauptverfasser:	Abonyo, B. O, Alexander, M. S, Heiman, A. S
Format:	Artikel
Sprache:	eng
Schlagworte:	Cell Line Chemokine CCL11 Chemokine CCL24 Chemokine CCL26 Chemokines, CC - biosynthesis Chemokines, CC - genetics Chemokines, CC - metabolism Eosinophils Epithelial Cells - metabolism Epithelial Cells - pathology Homeostasis Humans Interleukin-13 - pharmacology Interleukin-4 - pharmacology Pneumonia - metabolism Pneumonia - pathology Pneumonia - physiopathology Protein Biosynthesis Pulmonary Alveoli - metabolism Pulmonary Alveoli - pathology Pulmonary Alveoli - physiopathology Receptors, CCR3 Receptors, Chemokine - antagonists & inhibitors Receptors, Chemokine - genetics Receptors, Chemokine - metabolism Transcription, Genetic
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