Ciglitizone inhibits cell proliferation in human uterine leiomyoma via activation of store-operated Ca2+ channels

1 Department of Physiology, 2 Department of Obstetrics and Gynecology, and 3 Chronic Disease Research Center, Keimyung University School of Medicine, Daegu, Korea; and 4 Department of Oncology, Lombardi Cancer Center, Georgetown University, Washington, District of Columbia Submitted 23 March 2004 ;...

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Veröffentlicht in:	American Journal of Physiology: Cell Physiology 2005-02, Vol.288 (2), p.C389-C395
Hauptverfasser:	Kim, Byoung Ywong, Cho, Chi-Heum, Song, Dae-Kyu, Mun, Kyo-Cheol, Suh, Seong-Il, Kim, Sang-Pyo, Shin, Dong-Hoon, Jang, Byeong-Churl, Kwon, Taeg Kyu, Cha, Soon-Do, Bae, Insoo, Bae, Jae Hoon
Format:	Artikel
Sprache:	eng
Schlagworte:	Calcium - analysis Calcium - metabolism Calcium Channels - drug effects Calcium Channels - physiology Cell Proliferation - drug effects Cells, Cultured Dose-Response Relationship, Drug Female Humans Hypoglycemic Agents - pharmacology Intracellular Fluid - chemistry Intracellular Fluid - metabolism Leiomyoma - metabolism Myometrium - drug effects Myometrium - metabolism PPAR alpha - metabolism Sarcoplasmic Reticulum - metabolism Thiazolidinediones - pharmacology Uterine Neoplasms - metabolism
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