Stress impairs alpha(1A) adrenoceptor-mediated noradrenergic facilitation of GABAergic transmission in the basolateral amygdala

Intense or chronic stress can produce pathophysiological alterations in the systems involved in the stress response. The amygdala is a key component of the brain's neuronal network that processes and assigns emotional value to life's experiences, consolidates the memory of emotionally sign...

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Veröffentlicht in:	Neuropsychopharmacology (New York, N.Y.) N.Y.), 2004-01, Vol.29 (1), p.45
Hauptverfasser:	Braga, Maria Fatima M, Aroniadou-Anderjaska, Vassiliki, Manion, Sean T, Hough, Christopher J, Li, He
Format:	Artikel
Sprache:	eng
Schlagworte:	2-Amino-5-phosphonovalerate - pharmacology 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology Adrenergic alpha-Antagonists - pharmacology Adrenergic beta-Antagonists - pharmacology Aging Amygdala - cytology Amygdala - drug effects Amygdala - metabolism Amygdala - physiopathology Anesthetics, Local - pharmacology Animals Animals, Newborn Bicuculline - pharmacology Body Weight Dose-Response Relationship, Drug Drug Interactions Estrenes - pharmacology Excitatory Amino Acid Antagonists GABA Antagonists - pharmacology gamma-Aminobutyric Acid - metabolism Imidazoles - pharmacology In Vitro Techniques Isoquinolines - metabolism Male Membrane Potentials - drug effects Morpholines - pharmacology Neural Inhibition - drug effects Neurons - drug effects Neurons - physiology Norepinephrine - pharmacology Patch-Clamp Techniques - methods Phosphodiesterase Inhibitors - pharmacology Propranolol - pharmacology Pyrrolidinones - pharmacology Rats Rats, Sprague-Dawley Receptors, Adrenergic, alpha-1 - metabolism Restraint, Physical - methods Stress, Physiological - metabolism Tetrahydronaphthalenes - pharmacology Tetrodotoxin - pharmacology Time Factors
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