Role of iron in tumor cell protection from the pro-apoptotic effect of nitric oxide

F2The host defense against tumor cells is in part based upon the production of nitric oxide (NO) by activated macrophages. However, carcinogenesis may involve mechanisms that protect tumor cells from NO-mediated apoptosis. In the present study, we have assessed the effects of exogenous NO on the pro...

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Veröffentlicht in:	Cancer research (Chicago, Ill.) Ill.), 2001-07, Vol.61 (13), p.5289-5294
Hauptverfasser:	FEGER, Fredéric, FERRY-DUMAZET, Hélène, REIFFERS, Josy, MOSSALAYI, M. Djavad, MATSUDA, Maria Mamani, BORDENAVE, Joëlle, DUPOUY, Maryse, NUSSLER, Andreas K, AROCK, Michel, DEVEVEY, Lionel, NAFZIGER, Joëlle, GUILLOSSON, Jean-Jacques
Format:	Artikel
Sprache:	eng
Schlagworte:	Apoptosis - drug effects Apoptosis - physiology Biological and medical sciences Carcinogenesis, carcinogens and anticarcinogens Cell Cycle - drug effects Chemical agents Ferricyanides - pharmacology Humans Iron - metabolism Iron - physiology Medical sciences Neoplasms - metabolism Neoplasms - pathology Nitric Oxide - pharmacology Nitric Oxide Donors - pharmacology Penicillamine - analogs & derivatives Penicillamine - pharmacology S-Nitroso-N-Acetylpenicillamine Signal Transduction - physiology Tumor Cells, Cultured - drug effects Tumors
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