Basic FGF reduces stunning via a NOS2-dependent pathway in coronary-perfused mouse hearts

1 Cardiovascular Division and 4 Angiogenesis Research Center, Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts 02215; 2 Department of Cardiology, Medical University, D-30625 Hannover, Germany; and 3 Department of Surgery University of...

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Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 2000-07, Vol.279 (1), p.H260-H268
Hauptverfasser:	Hampton, Thomas G, Amende, Ivo, Fong, Jason, Laubach, Victor E, Li, Jian, Metais, Carolyn, Simons, Michael
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Calcium - metabolism Coronary Vessels - physiology Coronary Vessels - physiopathology Enzyme Inhibitors - pharmacology Female Fibroblast Growth Factor 2 - pharmacology Heart - drug effects Heart - physiology Heart - physiopathology In Vitro Techniques Lysine - analogs & derivatives Lysine - pharmacology Male Mice Mice, Inbred C57BL Mice, Knockout Myocardial Ischemia - physiopathology Myocardial Reperfusion Myocardial Stunning - drug therapy Myocardial Stunning - physiopathology NG-Nitroarginine Methyl Ester - pharmacology Nitric Oxide Synthase - deficiency Nitric Oxide Synthase - genetics Nitric Oxide Synthase - metabolism Nitric Oxide Synthase Type II Recombinant Proteins - pharmacology
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