Opposing roles of glucocorticoid receptor and mineralocorticoid receptor in trimethyltin-induced cytotoxicity in the mouse hippocampus

► TMT-mediated neurotoxicity is exacerbated by a type I corticosterone receptor (mineralocorticoid receptor) agonist. ► TMT-mediated neurotoxicity is ameliorated by a type I corticosterone receptor (mineralocorticoid receptor) antagonist. ► TMT neurotoxicity is exacerbated by a type II corticosteron...

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Veröffentlicht in:	Neuroscience letters 2012-03, Vol.511 (2), p.116-119
Hauptverfasser:	Ogita, Kiyokazu, Sugiyama, Chie, Acosta, Gabriela Beatriz, Kuramoto, Nobuyuki, Shuto, Makoto, Yoneyama, Masanori, Nakamura, Yukary, Shiba, Tatsuo, Yamaguchi, Taro
Format:	Artikel
Sprache:	eng
Schlagworte:	Aldosterone - pharmacology Animals Corticosterone - blood Glucocorticoid receptor hippocampus Hippocampus - drug effects Hippocampus - metabolism Mice Mineralocorticoid receptor Mineralocorticoid Receptor Antagonists - pharmacology Receptors, Glucocorticoid - metabolism Receptors, Glucocorticoid - physiology Receptors, Mineralocorticoid - agonists Receptors, Mineralocorticoid - metabolism Receptors, Mineralocorticoid - physiology Spironolactone - pharmacology Trimethyltin Trimethyltin Compounds - toxicity
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