Flavonoid combinations cause synergistic inhibition of proinflammatory mediator secretion from lipopolysaccharide-induced RAW 264.7 cells

Objectives We evaluated several flavonoid combinations for synergy in the inhibition of proinflammatory mediator synthesis in the RAW 264.7 cellular model of inflammation. Methods The inhibitory effect of chrysin, kaempferol, morin, silibinin, quercetin, diosmin and hesperidin upon nitric oxide (NO)...

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Veröffentlicht in:	Inflammation research 2010-09, Vol.59 (9), p.711-721
Hauptverfasser:	Harasstani, Omar A, Moin, Saidi, Tham, Chau Ling, Liew, Choi Yi, Ismail, Norazren, Rajajendram, Revathee, Harith, Hanis H, Zakaria, Zainul A, Mohamad, Azam S, Sulaiman, Mohamad R, Israf, Daud A
Format:	Artikel
Sprache:	eng
Schlagworte:	Allergology Animals Biomedical and Life Sciences Biomedicine Cell Line Dermatology Dinoprostone - antagonists & inhibitors Dinoprostone - metabolism Dose-Response Relationship, Drug Drug Synergism Flavonoid Flavonoids - pharmacology Flavonoids - therapeutic use Immunology Inflammation - drug therapy Inflammation - metabolism Isobologram Lipopolysaccharides - toxicity Macrophages - drug effects Mice Neurology nitric oxide Nitric Oxide - antagonists & inhibitors Nitric Oxide - metabolism Original Research Paper Pharmacology/Toxicology Prostaglandin E RAW 264.7 Rheumatology Synergy Tumor Necrosis Factor-alpha - antagonists & inhibitors Tumor Necrosis Factor-alpha - metabolism Tumour necrosis factor-α
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