Obesity induced by neonatal treatment with monosodium glutamate impairs microvascular reactivity in adult rats: Role of NO and prostanoids

Abstract Background and aim given that obesity is an independent risk factor for the development of cardiovascular diseases we decided to investigate the mechanisms involved in microvascular dysfunction using a monosodium glutamate (MSG)-induced model of obesity, which allows us to work on both norm...

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Veröffentlicht in:	Nutrition, metabolism, and cardiovascular diseases metabolism, and cardiovascular diseases, 2011-10, Vol.21 (10), p.808-816
Hauptverfasser:	Lobato, N.S, Filgueira, F.P, Akamine, E.H, Davel, A.P.C, Rossoni, L.V, Tostes, R.C, Carvalho, M.H.C, Fortes, Z.B
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Animals, Newborn Arterioles - enzymology Arterioles - metabolism Cardiovascular Cyclooxygenase 2 - analysis Male Mesenteric arteriolar bed Mesentery - blood supply Microvessels - physiopathology Monosodium glutamate Nitric oxide Nitric Oxide - physiology Nitric Oxide Synthase Type III - analysis Obesity Obesity - chemically induced Obesity - physiopathology Prostaglandins - physiology Prostanoids Rats Rats, Wistar Reactive oxygen species Reactive Oxygen Species - metabolism Sodium Glutamate - administration & dosage
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