Anti-Inflammatory Benefits of Antibiotic-Induced Neutrophil Apoptosis: Tulathromycin Induces Caspase-3-Dependent Neutrophil Programmed Cell Death and Inhibits NF-κB Signaling and CXCL8 Transcription

Anti-Inflammatory Benefits of Antibiotic-Induced Neutrophil Apoptosis: Tulathromycin Induces Caspase-3-Dependent Neutrophil Programmed Cell Death and Inhibits NF-κB Signaling and CXCL8 Transcription

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Veröffentlicht in:Antimicrobial Agents and Chemotherapy 2011-01, Vol.55 (1), p.338-348
Hauptverfasser: Fischer, Carrie D, Beatty, Jennifer K, Zvaigzne, Cheryl G, Morck, Douglas W, Lucas, Merlyn J, Buret, A. G
Format: Artikel
Sprache:eng
Schlagworte:
Alveoli
Anti-Bacterial Agents
Anti-Inflammatory Agents
Apoptosis
Caspase 3
Disaccharides
Experimental Therapeutics
Heterocyclic Compounds
Interleukin-8
Mannheimia haemolytica
Neutrophils
NF-kappa B
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container_end_page 348
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container_title Antimicrobial Agents and Chemotherapy
container_volume 55
creator Fischer, Carrie D
Beatty, Jennifer K
Zvaigzne, Cheryl G
Morck, Douglas W
Lucas, Merlyn J
Buret, A. G
description Classifications Services AAC Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit StumbleUpon Twitter current issue AAC About AAC Subscribers Authors Reviewers Advertisers Inquiries from the Press Permissions & Commercial Reprints ASM Journals Public Access Policy AAC RSS Feeds 1752 N Street N.W. • Washington DC 20036 202.737.3600 • 202.942.9355 fax • journals@asmusa.org Print ISSN: 0066-4804 Online ISSN: 1098-6596 Copyright © 2014 by the American Society for Microbiology.   For an alternate route to AAC .asm.org, visit: AAC       
doi_str_mv 10.1128/AAC.01052-10
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source EZB-FREE-00999 freely available EZB journals; PubMed Central
subjects Alveoli
Anti-Bacterial Agents
Anti-Inflammatory Agents
Apoptosis
Caspase 3
Disaccharides
Experimental Therapeutics
Heterocyclic Compounds
Interleukin-8
Mannheimia haemolytica
Neutrophils
NF-kappa B
title Anti-Inflammatory Benefits of Antibiotic-Induced Neutrophil Apoptosis: Tulathromycin Induces Caspase-3-Dependent Neutrophil Programmed Cell Death and Inhibits NF-κB Signaling and CXCL8 Transcription
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