An early treatment with 17‐β‐estradiol is neuroprotective against the long‐term effects of neonatal ionizing radiation exposure

J. Neurochem. (2011) 118, 626–635. Ionizing radiations can induce oxidative stress on target tissues, acting mainly through reactive oxygen species (ROS). The aim of this work was to investigate if 17‐β‐estradiol (βE) was able to prevent hippocampal‐related behavioral and biochemical changes induced...

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Veröffentlicht in:	Journal of neurochemistry 2011-08, Vol.118 (4), p.626-635
Hauptverfasser:	Caceres, Lucila G., Uran, Soledad L., Zorrilla Zubilete, María A., Romero, Juan I., Capani, Francisco, Guelman, Laura R.
Format:	Artikel
Sprache:	eng
Schlagworte:	17‐β‐estradiol Animals Animals, Newborn Antioxidants Antioxidants - metabolism Anxiety Anxiety - psychology Avoidance Learning - drug effects Avoidance Learning - radiation effects behavior Biological and medical sciences Central nervous system Electrophysiology Estradiol - pharmacology Female Fundamental and applied biological sciences. Psychology Habituation Habituation, Psychophysiologic - drug effects Habituation, Psychophysiologic - radiation effects Hippocampus Hippocampus - drug effects Hippocampus - metabolism Hippocampus - radiation effects Ionizing radiation Long-term effects Male Medical sciences Memory Memory - drug effects Motor Activity - drug effects Motor Activity - radiation effects Neonates Neuropharmacology Neuroprotection Neuroprotective agent Neuroprotective Agents Oxidative stress Pharmacology. Drug treatments PKC Pregnancy Protein kinase C Protein Kinase C - metabolism Radiation Injuries, Experimental - prevention & control Radiation-Protective Agents Rats Reactive oxygen species Reactive Oxygen Species - metabolism Reactive Oxygen Species - radiation effects ROS Time Vertebrates: nervous system and sense organs X-Rays
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