Effects of imidacloprid exposure on Chironomus riparius Meigen larvae: Linking acetylcholinesterase activity to behaviour

Imidacloprid (IMI) is an insecticide that interferes with the transmission of stimuli in the nervous system of insects. It is neurotoxic by mimicking nicotine through its binding to the nicotinic acetylcholine receptor. In this work, experiments comprising 96 h exposure followed by 48 h in clean med...

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Veröffentlicht in:	Ecotoxicology and environmental safety 2011-07, Vol.74 (5), p.1210-1215
Hauptverfasser:	Azevedo-Pereira, H.M.V.S., Lemos, M.F.L., Soares, A.M.V.M.
Format:	Artikel
Sprache:	eng
Schlagworte:	acetylcholinesterase Acetylcholinesterase - metabolism Animal, plant and microbial ecology Animals Applied ecology Behavior, Animal - drug effects Behavioural Behavioural parameters Biochemistry Biological and medical sciences Chironomidae - drug effects Chironomidae - metabolism Chironomidae - physiology Chironomus riparius cholinergic receptors Cleaning Dose-Response Relationship, Drug Ecotoxicology Ecotoxicology, biological effects of pollution Fundamental and applied biological sciences. Psychology General aspects Imidacloprid Imidazoles - toxicity Insecticides - toxicity insects Joining Larva - drug effects Larva - metabolism larvae Linking Linking biomarkers locomotion mechanism of action Motor Activity - drug effects Neonicotinoids nervous system Nervous System - drug effects Neurotoxicity nicotine Nitro Compounds - toxicity Recovery Techniques Ventilation xenobiotics
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