The etiologic classification of epilepsy
Summary The etiology of epilepsy is a major determinant of clinical course and prognosis, yet the current classifications of epilepsy do not list etiology in any detail. In this article, a classification (database) of the etiologies of epilepsy is proposed. In this scheme, the etiology of epilepsy i...
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| Veröffentlicht in: | Epilepsia (Copenhagen) 2011-06, Vol.52 (6), p.1052-1057 |
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| container_title | Epilepsia (Copenhagen) |
| container_volume | 52 |
| creator | Shorvon, Simon D. |
| description | Summary
The etiology of epilepsy is a major determinant of clinical course and prognosis, yet the current classifications of epilepsy do not list etiology in any detail. In this article, a classification (database) of the etiologies of epilepsy is proposed. In this scheme, the etiology of epilepsy is divided into four categories: idiopathic, symptomatic, provoked, and cryptogenic. These are defined and subcategories are proposed. A commentary addressing the following points is included: problems associated with assigning causation, symptomatic versus idiopathic epilepsy, focal versus generalized epilepsy, acquired epilepsy, acute symptomatic epilepsy, risk factor analysis, provoked epilepsy genetic and developmental epilepsy, and epilepsy as a disease not a symptom. |
| doi_str_mv | 10.1111/j.1528-1167.2011.03041.x |
| format | Article |
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The etiology of epilepsy is a major determinant of clinical course and prognosis, yet the current classifications of epilepsy do not list etiology in any detail. In this article, a classification (database) of the etiologies of epilepsy is proposed. In this scheme, the etiology of epilepsy is divided into four categories: idiopathic, symptomatic, provoked, and cryptogenic. These are defined and subcategories are proposed. A commentary addressing the following points is included: problems associated with assigning causation, symptomatic versus idiopathic epilepsy, focal versus generalized epilepsy, acquired epilepsy, acute symptomatic epilepsy, risk factor analysis, provoked epilepsy genetic and developmental epilepsy, and epilepsy as a disease not a symptom.</description><identifier>ISSN: 0013-9580</identifier><identifier>EISSN: 1528-1167</identifier><identifier>DOI: 10.1111/j.1528-1167.2011.03041.x</identifier><identifier>PMID: 21449936</identifier><identifier>CODEN: EPILAK</identifier><language>eng</language><publisher>Oxford, UK: Blackwell Publishing Ltd</publisher><subject>Animals ; Anticonvulsants. Antiepileptics. Antiparkinson agents ; Biological and medical sciences ; Cause ; Classification ; Epilepsy ; Epilepsy - classification ; Epilepsy - etiology ; Epilepsy - physiopathology ; Etiology ; Headache. Facial pains. Syncopes. Epilepsia. Intracranial hypertension. Brain oedema. Cerebral palsy ; Humans ; Idiopathic epilepsy ; Medical sciences ; Nervous system (semeiology, syndromes) ; Neurology ; Neuropharmacology ; Pharmacology. Drug treatments ; Prognosis ; Provoked epilepsy ; Risk Factors ; Symptomatic epilepsy</subject><ispartof>Epilepsia (Copenhagen), 2011-06, Vol.52 (6), p.1052-1057</ispartof><rights>Wiley Periodicals, Inc. © 2011 International League Against Epilepsy</rights><rights>2015 INIST-CNRS</rights><rights>Wiley Periodicals, Inc. © 2011 International League Against Epilepsy.</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c5081-f13022ece51cd73098bebf4aa1bbba48554ca43669affa310efae185ac62993</citedby><cites>FETCH-LOGICAL-c5081-f13022ece51cd73098bebf4aa1bbba48554ca43669affa310efae185ac62993</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://onlinelibrary.wiley.com/doi/pdf/10.1111%2Fj.1528-1167.2011.03041.x$$EPDF$$P50$$Gwiley$$H</linktopdf><linktohtml>$$Uhttps://onlinelibrary.wiley.com/doi/full/10.1111%2Fj.1528-1167.2011.03041.x$$EHTML$$P50$$Gwiley$$H</linktohtml><link.rule.ids>314,776,780,1411,1427,27903,27904,45553,45554,46387,46811</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=24339409$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/21449936$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Shorvon, Simon D.</creatorcontrib><title>The etiologic classification of epilepsy</title><title>Epilepsia (Copenhagen)</title><addtitle>Epilepsia</addtitle><description>Summary
The etiology of epilepsy is a major determinant of clinical course and prognosis, yet the current classifications of epilepsy do not list etiology in any detail. In this article, a classification (database) of the etiologies of epilepsy is proposed. In this scheme, the etiology of epilepsy is divided into four categories: idiopathic, symptomatic, provoked, and cryptogenic. These are defined and subcategories are proposed. A commentary addressing the following points is included: problems associated with assigning causation, symptomatic versus idiopathic epilepsy, focal versus generalized epilepsy, acquired epilepsy, acute symptomatic epilepsy, risk factor analysis, provoked epilepsy genetic and developmental epilepsy, and epilepsy as a disease not a symptom.</description><subject>Animals</subject><subject>Anticonvulsants. Antiepileptics. Antiparkinson agents</subject><subject>Biological and medical sciences</subject><subject>Cause</subject><subject>Classification</subject><subject>Epilepsy</subject><subject>Epilepsy - classification</subject><subject>Epilepsy - etiology</subject><subject>Epilepsy - physiopathology</subject><subject>Etiology</subject><subject>Headache. Facial pains. Syncopes. Epilepsia. Intracranial hypertension. Brain oedema. Cerebral palsy</subject><subject>Humans</subject><subject>Idiopathic epilepsy</subject><subject>Medical sciences</subject><subject>Nervous system (semeiology, syndromes)</subject><subject>Neurology</subject><subject>Neuropharmacology</subject><subject>Pharmacology. Drug treatments</subject><subject>Prognosis</subject><subject>Provoked epilepsy</subject><subject>Risk Factors</subject><subject>Symptomatic epilepsy</subject><issn>0013-9580</issn><issn>1528-1167</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2011</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqNkFFLwzAQgIMobk7_ghRE9KX1Lknb9MEHGVMHAwX3HtIs0ZZunc2K2783dXOCL3ovOZLvcncfIQFChD5uyghjKkLEJI0oIEbAgGO0PiD9_cMh6QMgC7NYQI-cOFcCQJqk7Jj0KHKeZSzpk-vpmwnMqqir-rXQga6Uc4UttPJXi6C2gVkWlVm6zSk5sqpy5mx3DsjL_Wg6fAwnTw_j4d0k1DEIDC0yoNRoE6OepQwykZvccqUwz3PFRRxzrThLkkxZqxiCscqgiJVOqJ9oQK62vy6b-r01biXnhdOmqtTC1K2TQjDADBP-N5lSpAmwjrz4RZZ12yz8EhJjTCkTlDNPiS2lm9q5xli5bIq5ajYSQXbSZSk7t7JzKzvp8ku6XPvS812DNp-b2b7w27IHLneAclpVtlELXbgfzrfPOHTb3265D-988-8B5Oh53GXsE7JYmrA</recordid><startdate>201106</startdate><enddate>201106</enddate><creator>Shorvon, Simon D.</creator><general>Blackwell Publishing Ltd</general><general>Wiley-Blackwell</general><general>Wiley Subscription Services, Inc</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7TK</scope><scope>7X8</scope></search><sort><creationdate>201106</creationdate><title>The etiologic classification of epilepsy</title><author>Shorvon, Simon D.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c5081-f13022ece51cd73098bebf4aa1bbba48554ca43669affa310efae185ac62993</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2011</creationdate><topic>Animals</topic><topic>Anticonvulsants. Antiepileptics. Antiparkinson agents</topic><topic>Biological and medical sciences</topic><topic>Cause</topic><topic>Classification</topic><topic>Epilepsy</topic><topic>Epilepsy - classification</topic><topic>Epilepsy - etiology</topic><topic>Epilepsy - physiopathology</topic><topic>Etiology</topic><topic>Headache. Facial pains. Syncopes. Epilepsia. Intracranial hypertension. Brain oedema. Cerebral palsy</topic><topic>Humans</topic><topic>Idiopathic epilepsy</topic><topic>Medical sciences</topic><topic>Nervous system (semeiology, syndromes)</topic><topic>Neurology</topic><topic>Neuropharmacology</topic><topic>Pharmacology. Drug treatments</topic><topic>Prognosis</topic><topic>Provoked epilepsy</topic><topic>Risk Factors</topic><topic>Symptomatic epilepsy</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Shorvon, Simon D.</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>Neurosciences Abstracts</collection><collection>MEDLINE - Academic</collection><jtitle>Epilepsia (Copenhagen)</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Shorvon, Simon D.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>The etiologic classification of epilepsy</atitle><jtitle>Epilepsia (Copenhagen)</jtitle><addtitle>Epilepsia</addtitle><date>2011-06</date><risdate>2011</risdate><volume>52</volume><issue>6</issue><spage>1052</spage><epage>1057</epage><pages>1052-1057</pages><issn>0013-9580</issn><eissn>1528-1167</eissn><coden>EPILAK</coden><abstract>Summary
The etiology of epilepsy is a major determinant of clinical course and prognosis, yet the current classifications of epilepsy do not list etiology in any detail. In this article, a classification (database) of the etiologies of epilepsy is proposed. In this scheme, the etiology of epilepsy is divided into four categories: idiopathic, symptomatic, provoked, and cryptogenic. These are defined and subcategories are proposed. A commentary addressing the following points is included: problems associated with assigning causation, symptomatic versus idiopathic epilepsy, focal versus generalized epilepsy, acquired epilepsy, acute symptomatic epilepsy, risk factor analysis, provoked epilepsy genetic and developmental epilepsy, and epilepsy as a disease not a symptom.</abstract><cop>Oxford, UK</cop><pub>Blackwell Publishing Ltd</pub><pmid>21449936</pmid><doi>10.1111/j.1528-1167.2011.03041.x</doi><tpages>6</tpages><oa>free_for_read</oa></addata></record> |
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| ispartof | Epilepsia (Copenhagen), 2011-06, Vol.52 (6), p.1052-1057 |
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| language | eng |
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| source | MEDLINE; Wiley Online Library Journals Frontfile Complete; Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals; Wiley Free Content; IngentaConnect Free/Open Access Journals; Alma/SFX Local Collection |
| subjects | Animals Anticonvulsants. Antiepileptics. Antiparkinson agents Biological and medical sciences Cause Classification Epilepsy Epilepsy - classification Epilepsy - etiology Epilepsy - physiopathology Etiology Headache. Facial pains. Syncopes. Epilepsia. Intracranial hypertension. Brain oedema. Cerebral palsy Humans Idiopathic epilepsy Medical sciences Nervous system (semeiology, syndromes) Neurology Neuropharmacology Pharmacology. Drug treatments Prognosis Provoked epilepsy Risk Factors Symptomatic epilepsy |
| title | The etiologic classification of epilepsy |
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