Bone formation markers in patients with glucocorticoid-induced osteoporosis treated with teriparatide or alendronate

Abstract Reduced osteoblast function is a primary defect in glucocorticoid-induced osteoporosis (GIO), and is reflected by decreased biochemical markers of bone formation, such as serum osteocalcin (OC) and procollagen type I N-terminal propeptide (PINP). This analysis compared the effects of teripa...

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Veröffentlicht in:	Bone (New York, N.Y.) N.Y.), 2010-04, Vol.46 (4), p.929-934
Hauptverfasser:	Eastell, Richard, Chen, Peiqi, Saag, Kenneth G, Burshell, Alan L, Wong, Mayme, Warner, Margaret R, Krege, John H
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Aged Alendronate - therapeutic use Biomarkers - blood Bone Density - drug effects Bone Density - physiology Bone Density Conservation Agents - therapeutic use Chi-Square Distribution Double-Blind Method Female Glucocorticoids - adverse effects Humans Male Middle Aged Orthopedics Osteocalcin - blood Osteogenesis - drug effects Osteogenesis - physiology Osteoporosis - chemically induced Osteoporosis - drug therapy Osteoporosis - metabolism Peptide Fragments - blood Procollagen - blood Teriparatide - therapeutic use Treatment Outcome
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