North American ginseng protects the heart from ischemia and reperfusion injury via upregulation of endothelial nitric oxide synthase

[Display omitted] ► Role of endothelial nitric oxide synthase (eNOS) in the cardioprotection of ginseng was studied. ► Ginseng increased eNOS expression and inhibited anoxia/reoxygenation (A/R)-induced apoptosis in cultured cardiomyocytes. ► In vivo ginseng treatment increased myocardial eNOS expres...

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Veröffentlicht in:	Pharmacological research 2011-09, Vol.64 (3), p.195-202
Hauptverfasser:	Wu, Yan, Lu, Xiangru, Xiang, Fu-Li, Lui, Edmund M.K., Feng, Qingping
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apoptosis - drug effects Cardioprotection Cardiotonic Agents - isolation & purification Cardiotonic Agents - therapeutic use Cells, Cultured Endothelial nitric oxide synthase Gene Deletion Ginseng Heart - drug effects Mice Mice, Inbred C57BL Myocardial ischemia and reperfusion Myocardial Reperfusion Injury - enzymology Myocardial Reperfusion Injury - pathology Myocardial Reperfusion Injury - prevention & control Myocytes, Cardiac - drug effects Myocytes, Cardiac - enzymology Myocytes, Cardiac - pathology Nitric Oxide Synthase Type III - genetics North America Panax - chemistry Phosphorylation - drug effects Phytotherapy Plant Preparations - isolation & purification Plant Preparations - therapeutic use Proto-Oncogene Proteins c-akt - metabolism Up-Regulation - drug effects
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