Cardiac dysfunction induced by experimental myocardial infarction impairs the host defense response to bacterial infection in mice because of reduced phagocytosis of Kupffer cells

Objective This study was undertaken to investigate the effects of cardiac dysfunction induced by experimental myocardial infarction on the host defense response to bacterial infection and the role of Kupffer cells in mediating this response. Methods Myocardial infarction was induced in C57BL/6 mice...

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Veröffentlicht in:	The Journal of thoracic and cardiovascular surgery 2010-09, Vol.140 (3), p.624-632.e3
Hauptverfasser:	Nogami, Yashiro, MD, Kinoshita, Manabu, MD, Takase, Bonpei, MD, Inatsu, Akihito, MD, Ishihara, Masayuki, PhD, Seki, Shuhji, MD, Maehara, Tadaaki, MD
Format:	Artikel
Sprache:	eng
Schlagworte:	Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy Animals Biological and medical sciences C-reactive protein C-Reactive Protein - administration & dosage Cardiology. Vascular system Cardiothoracic Surgery Cells, Cultured Coronary heart disease Disease Models, Animal Escherichia coli Escherichia coli Infections - immunology Heart Immunity, Innate Immunoglobulin M - administration & dosage Immunoglobulin M - metabolism Inflammation Mediators - blood Injections, Intraperitoneal Interferon-gamma - blood Interleukin-18 - administration & dosage Kupffer Cells - immunology Kupffer Cells - microbiology Male Medical sciences Mice Mice, Inbred C57BL Myocardial Infarction - diagnostic imaging Myocardial Infarction - immunology Myocardial Infarction - physiopathology Myocarditis. Cardiomyopathies Phagocytosis Pneumology Time Factors Tumor Necrosis Factor-alpha - blood Ultrasonography Ventricular Dysfunction, Left - diagnostic imaging Ventricular Dysfunction, Left - immunology Ventricular Dysfunction, Left - physiopathology Ventricular Function, Left
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