Alterations of GABA A and glutamate receptor subunits and heat shock protein in rat hippocampus following traumatic brain injury and in posttraumatic epilepsy

Traumatic brain injury (TBI) can result in the development of posttraumatic epilepsy (PTE). Recently, we reported differential alterations in tonic and phasic GABA A receptor (GABA AR) currents in hippocampal dentate granule cells 90 days after controlled cortical impact (CCI) ( Mtchedlishvili et al...

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Veröffentlicht in:	Epilepsy research 2011-06, Vol.95 (1), p.20-34
Hauptverfasser:	Kharlamov, Elena A., Lepsveridze, Eka, Meparishvili, Maia, Solomonia, Revaz O., Lu, Bo, Miller, Eric R., Kelly, Kevin M., Mtchedlishvili, Zakaria
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Brain Injuries - genetics Brain Injuries - metabolism Controlled cortical impact Epilepsy, Post-Traumatic - genetics Epilepsy, Post-Traumatic - metabolism Epileptogenesis Gene Expression Regulation Hippocampus Hippocampus - injuries Hippocampus - metabolism HSP70 Heat-Shock Proteins - biosynthesis HSP70 Heat-Shock Proteins - genetics HSP90 Heat-Shock Proteins - biosynthesis HSP90 Heat-Shock Proteins - genetics Male Nerve Tissue Proteins - biosynthesis Nerve Tissue Proteins - genetics Posttraumatic epilepsy Rats Rats, Sprague-Dawley Receptors, AMPA - biosynthesis Receptors, AMPA - genetics Receptors, GABA-A - biosynthesis Receptors, GABA-A - genetics Receptors, N-Methyl-D-Aspartate - biosynthesis Receptors, N-Methyl-D-Aspartate - genetics Traumatic brain injury Western blot
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