Silencing of ZnT1 reduces Zn super(2+) efflux in cultured cortical neurons

Zinc dyshomeostasis in brain might be involved in the pathogenesis of a series of brain diseases such as Alzheimer's disease and stroke. It is essential that the level of intracellular free Zn super(2+) in neurons is tightly controlled to maintain a narrow window of optimal concentration. The p...

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Veröffentlicht in:	Neuroscience letters 2009-01, Vol.450 (2), p.206-210
Hauptverfasser:	Qin, Yan, Thomas, Dustin, Fontaine, Charles P, Colvin, Robert A
Format:	Artikel
Sprache:	eng
Schlagworte:	Alzheimer's disease
Online-Zugang:	Volltext
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