Altered host response to murine gammaherpesvirus 68 infection in mice lacking the tachykinin 1 gene and the receptor for substance P

Abstract The tachykinins are implicated in neurogenic inflammation and the neuropeptide substance P in particular has been shown to be a proinflammatory mediator. A role for the tachykinins in host response to viral infection has been previously demonstrated using either TAC1- or NK1 receptor-defici...

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Veröffentlicht in:	Neuropeptides (Edinburgh) 2011-02, Vol.45 (1), p.49-53
Hauptverfasser:	Quinn, John P, Kipar, Anja, Hughes, David J, Bennett, Elaine, Cox, Helen, McLaughlin, Lynn, Zimmer, Andreas, Hunt, Stephen P, Stewart, James P
Format:	Artikel
Sprache:	eng
Schlagworte:	Advanced Basic Science Animals Biological and medical sciences Endocrinology & Metabolism Fundamental and applied biological sciences. Psychology Herpesviridae Infections - immunology Host-Pathogen Interactions Mice Mice, Inbred C57BL Mice, Knockout Murine gammaherpesvirus Murine gammaherpesvirus 68 NK1 Receptors, Neurokinin-1 - genetics Receptors, Neurokinin-1 - physiology Rhadinovirus - immunology Rhadinovirus - pathogenicity Substance P Substance P - immunology Tachykinin Tachykinins - genetics Tachykinins - physiology Transgenic mice Tumor Virus Infections - immunology Vertebrates: endocrinology Virus
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