Relaxin counteracts the altered gastric motility of dystrophic (mdx) mice: functional and immunohistochemical evidence for the involvement of nitric oxide

Impaired gastric motility ascribable to a defective nitric oxide (NO) production has been reported in dystrophic (mdx) mice. Since relaxin upregulates NO biosynthesis, its effects on the motor responses and NO synthase (NOS) expression in the gastric fundus of mdx mice were investigated. Mechanical...

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Veröffentlicht in:	American journal of physiology: endocrinology and metabolism 2011-02, Vol.300 (2), p.E380-E391
Hauptverfasser:	Vannucchi, M G, Garella, R, Cipriani, G, Baccari, M C
Format:	Artikel
Sprache:	eng
Schlagworte:	Actins - metabolism Animals Biochemistry Biosynthesis Blotting, Western Electric Stimulation Gastric Fundus Gastrointestinal Motility - drug effects Gastrointestinal Motility - genetics Immunohistochemistry In Vitro Techniques Isoenzymes - biosynthesis Isoenzymes - genetics Male Mice Mice, Inbred C57BL Mice, Inbred mdx Muscle Contraction - physiology Muscle Relaxation - drug effects Nitric oxide Nitric Oxide - metabolism Nitric Oxide - physiology Nitric Oxide Synthase - biosynthesis Nitric Oxide Synthase - genetics Relaxin - pharmacology Rodents Signal transduction Stomach Stomach - drug effects Stomach - enzymology
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