Nonuniform Recovery of Excitability in Ventricular Muscle
Recovery of excitability of ventricular muscle was measured at numerous points in exposed dog ventricles at varying distances along six radial axes from a primary point of stimulation. Temporal dispersion of recovery of excitability at various points equidistant from the point of stimulation was min...
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| Veröffentlicht in: | Circulation research 1964-01, Vol.14 (1), p.44-60 |
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| container_title | Circulation research |
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| creator | Han, Jaok Moe, Gordon K |
| description | Recovery of excitability of ventricular muscle was measured at numerous points in exposed dog ventricles at varying distances along six radial axes from a primary point of stimulation. Temporal dispersion of recovery of excitability at various points equidistant from the point of stimulation was minimal after a basic beat but was increased after an early premature beat. The degree of dispersion following a basic beat was increased by stimulation of the cardiac sympathetic nerves, administration of chloroform, ouabain intoxication, administration of higher doses of quinidine, myocardial ischemia, and hypothermia, but it was decreased by administration of sympathomimetic amines. |
| doi_str_mv | 10.1161/01.res.14.1.44 |
| format | Article |
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Temporal dispersion of recovery of excitability at various points equidistant from the point of stimulation was minimal after a basic beat but was increased after an early premature beat. The degree of dispersion following a basic beat was increased by stimulation of the cardiac sympathetic nerves, administration of chloroform, ouabain intoxication, administration of higher doses of quinidine, myocardial ischemia, and hypothermia, but it was decreased by administration of sympathomimetic amines.</description><identifier>ISSN: 0009-7330</identifier><identifier>EISSN: 1524-4571</identifier><identifier>DOI: 10.1161/01.res.14.1.44</identifier><identifier>PMID: 14104163</identifier><language>eng</language><publisher>United States: American Heart Association, Inc</publisher><subject>Animals ; Chloroform ; Dogs ; Electric Stimulation ; Electrophysiology ; Epinephrine ; Ganglia ; Ganglia, Autonomic ; Heart Conduction System ; Heart Ventricles ; Hypothermia ; Hypothermia, Induced ; Ischemia ; Myocardium ; Norepinephrine ; Old Medline ; Ouabain ; Quinidine ; Strophanthins ; Ventricular Fibrillation</subject><ispartof>Circulation research, 1964-01, Vol.14 (1), p.44-60</ispartof><rights>1964 American Heart Association, Inc.</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c4874-c52dd9a01e956fbf42e538bd0881ef0d594d75329118f454a01ab247ea4e96433</citedby></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,780,784,3687,27924,27925</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/14104163$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Han, Jaok</creatorcontrib><creatorcontrib>Moe, Gordon K</creatorcontrib><title>Nonuniform Recovery of Excitability in Ventricular Muscle</title><title>Circulation research</title><addtitle>Circ Res</addtitle><description>Recovery of excitability of ventricular muscle was measured at numerous points in exposed dog ventricles at varying distances along six radial axes from a primary point of stimulation. Temporal dispersion of recovery of excitability at various points equidistant from the point of stimulation was minimal after a basic beat but was increased after an early premature beat. The degree of dispersion following a basic beat was increased by stimulation of the cardiac sympathetic nerves, administration of chloroform, ouabain intoxication, administration of higher doses of quinidine, myocardial ischemia, and hypothermia, but it was decreased by administration of sympathomimetic amines.</description><subject>Animals</subject><subject>Chloroform</subject><subject>Dogs</subject><subject>Electric Stimulation</subject><subject>Electrophysiology</subject><subject>Epinephrine</subject><subject>Ganglia</subject><subject>Ganglia, Autonomic</subject><subject>Heart Conduction System</subject><subject>Heart Ventricles</subject><subject>Hypothermia</subject><subject>Hypothermia, Induced</subject><subject>Ischemia</subject><subject>Myocardium</subject><subject>Norepinephrine</subject><subject>Old Medline</subject><subject>Ouabain</subject><subject>Quinidine</subject><subject>Strophanthins</subject><subject>Ventricular Fibrillation</subject><issn>0009-7330</issn><issn>1524-4571</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1964</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNpFkMtLw0AQhxdRbH1cPUpO3hJnsrN5HEXqA6pCfVyXTTKh0W2iu4na_96UFjwNM3zz48cnxBlChJjgJWDk2EdIEUZEe2KKKqaQVIr7YgoAeZhKCRNx5P07AJKM80MxQUIgTORU5I9dO7RN3blVsOCy-2a3Dro6mP2WTW-Kxjb9Omja4I3b3jXlYI0LHgZfWj4RB7Wxnk9381i83sxeru_C-dPt_fXVPCwpSyksVVxVuQHkXCV1UVPMSmZFBVmGXEOlcqpSNdZCzGpSNJKmiCllQ5wnJOWxuNjmfrrua2Df61XjS7bWtNwNXmeSlMrSeASjLVi6znvHtf50zcq4tUbQG1kaUC9mzxpJoyYaH853yUOx4uof39kZAdoCP53t2fkPO_yw00s2tl_q0S5IwDjEsSfguIWbE8k_7rBzqw</recordid><startdate>196401</startdate><enddate>196401</enddate><creator>Han, Jaok</creator><creator>Moe, Gordon K</creator><general>American Heart Association, Inc</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>196401</creationdate><title>Nonuniform Recovery of Excitability in Ventricular Muscle</title><author>Han, Jaok ; Moe, Gordon K</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c4874-c52dd9a01e956fbf42e538bd0881ef0d594d75329118f454a01ab247ea4e96433</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1964</creationdate><topic>Animals</topic><topic>Chloroform</topic><topic>Dogs</topic><topic>Electric Stimulation</topic><topic>Electrophysiology</topic><topic>Epinephrine</topic><topic>Ganglia</topic><topic>Ganglia, Autonomic</topic><topic>Heart Conduction System</topic><topic>Heart Ventricles</topic><topic>Hypothermia</topic><topic>Hypothermia, Induced</topic><topic>Ischemia</topic><topic>Myocardium</topic><topic>Norepinephrine</topic><topic>Old Medline</topic><topic>Ouabain</topic><topic>Quinidine</topic><topic>Strophanthins</topic><topic>Ventricular Fibrillation</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Han, Jaok</creatorcontrib><creatorcontrib>Moe, Gordon K</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Circulation research</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Han, Jaok</au><au>Moe, Gordon K</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Nonuniform Recovery of Excitability in Ventricular Muscle</atitle><jtitle>Circulation research</jtitle><addtitle>Circ Res</addtitle><date>1964-01</date><risdate>1964</risdate><volume>14</volume><issue>1</issue><spage>44</spage><epage>60</epage><pages>44-60</pages><issn>0009-7330</issn><eissn>1524-4571</eissn><abstract>Recovery of excitability of ventricular muscle was measured at numerous points in exposed dog ventricles at varying distances along six radial axes from a primary point of stimulation. Temporal dispersion of recovery of excitability at various points equidistant from the point of stimulation was minimal after a basic beat but was increased after an early premature beat. The degree of dispersion following a basic beat was increased by stimulation of the cardiac sympathetic nerves, administration of chloroform, ouabain intoxication, administration of higher doses of quinidine, myocardial ischemia, and hypothermia, but it was decreased by administration of sympathomimetic amines.</abstract><cop>United States</cop><pub>American Heart Association, Inc</pub><pmid>14104163</pmid><doi>10.1161/01.res.14.1.44</doi><tpages>17</tpages><oa>free_for_read</oa></addata></record> |
| fulltext | fulltext |
| identifier | ISSN: 0009-7330 |
| ispartof | Circulation research, 1964-01, Vol.14 (1), p.44-60 |
| issn | 0009-7330 1524-4571 |
| language | eng |
| recordid | cdi_proquest_miscellaneous_83455872 |
| source | MEDLINE; American Heart Association Journals; Journals@Ovid Complete; EZB-FREE-00999 freely available EZB journals |
| subjects | Animals Chloroform Dogs Electric Stimulation Electrophysiology Epinephrine Ganglia Ganglia, Autonomic Heart Conduction System Heart Ventricles Hypothermia Hypothermia, Induced Ischemia Myocardium Norepinephrine Old Medline Ouabain Quinidine Strophanthins Ventricular Fibrillation |
| title | Nonuniform Recovery of Excitability in Ventricular Muscle |
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