Spontaneous ventricular parasystole initiating ventricular tachycardia
Ventricular parasystolic rhythms are rarely hemodynamically significant. However, ventricular parasystolic beats, like fixed rate ventricular pacing stimuli, have the theoretical potential to initiate repetitive beating when they fall in the vulnerable period. In the case presented here, spontaneous...
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Veröffentlicht in: | Journal of electrocardiology 1973, Vol.6 (1), p.63-70 |
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description | Ventricular parasystolic rhythms are rarely hemodynamically significant. However, ventricular parasystolic beats, like fixed rate ventricular pacing stimuli, have the theoretical potential to initiate repetitive beating when they fall in the vulnerable period. In the case presented here, spontaneously occurring ventricular parasystolic beats were able to initiate hemodynamically significant ventricular tachycardias. The ventricular tachycardia, however, did not disturb the parasystolic rhythm whose beats continued to occur as expected and were able to terminate the tachycardia. Thus, ventricular tachycardia could be terminated by a ventricular parasystolic beat which originated in the same focus as the ventricular parasystolic beat which initiated the tachycardia. |
doi_str_mv | 10.1016/S0022-0736(73)80026-3 |
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However, ventricular parasystolic beats, like fixed rate ventricular pacing stimuli, have the theoretical potential to initiate repetitive beating when they fall in the vulnerable period. In the case presented here, spontaneously occurring ventricular parasystolic beats were able to initiate hemodynamically significant ventricular tachycardias. The ventricular tachycardia, however, did not disturb the parasystolic rhythm whose beats continued to occur as expected and were able to terminate the tachycardia. Thus, ventricular tachycardia could be terminated by a ventricular parasystolic beat which originated in the same focus as the ventricular parasystolic beat which initiated the tachycardia.</description><identifier>ISSN: 0022-0736</identifier><identifier>EISSN: 1532-8430</identifier><identifier>DOI: 10.1016/S0022-0736(73)80026-3</identifier><identifier>PMID: 4691404</identifier><language>eng</language><publisher>United States: Elsevier Inc</publisher><subject>Aged ; Arrhythmias, Cardiac ; Electrocardiography ; Heart Conduction System - physiopathology ; Heart Failure - etiology ; Heart Rate ; Heart Ventricles - physiopathology ; Humans ; Male ; Pacemaker, Artificial ; Quinidine - therapeutic use ; Tachycardia - etiology ; Tachycardia - therapy</subject><ispartof>Journal of electrocardiology, 1973, Vol.6 (1), p.63-70</ispartof><rights>1973 Research in Electrocardiology, Inc. 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However, ventricular parasystolic beats, like fixed rate ventricular pacing stimuli, have the theoretical potential to initiate repetitive beating when they fall in the vulnerable period. In the case presented here, spontaneously occurring ventricular parasystolic beats were able to initiate hemodynamically significant ventricular tachycardias. The ventricular tachycardia, however, did not disturb the parasystolic rhythm whose beats continued to occur as expected and were able to terminate the tachycardia. Thus, ventricular tachycardia could be terminated by a ventricular parasystolic beat which originated in the same focus as the ventricular parasystolic beat which initiated the tachycardia.</description><subject>Aged</subject><subject>Arrhythmias, Cardiac</subject><subject>Electrocardiography</subject><subject>Heart Conduction System - physiopathology</subject><subject>Heart Failure - etiology</subject><subject>Heart Rate</subject><subject>Heart Ventricles - physiopathology</subject><subject>Humans</subject><subject>Male</subject><subject>Pacemaker, Artificial</subject><subject>Quinidine - therapeutic use</subject><subject>Tachycardia - etiology</subject><subject>Tachycardia - therapy</subject><issn>0022-0736</issn><issn>1532-8430</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1973</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqFkE1Lw0AQhhdRaq3-hEJPoofo7EeSzUmkWBUKHqrnZbs70ZU0ibubQv-96QcFT15mGOZ952UeQsYU7ijQ7H4BwFgCOc9ucn4r-ylL-AkZ0pSzRAoOp2R4lJyTixC-AaBgORuQgcgKKkAMyWzRNnXUNTZdmKyxjt6ZrtJ-0mqvwybEpsKJq110Orr6848kavO1Mdpbpy_JWamrgFeHPiIfs6f36Usyf3t-nT7OE8MziElRINcUQAvDBFJrl8JmMmUitWlaFoZmRlLQeblk2ghpKdK-MpRFnhYcDR-R6_3d1jc_HYaoVi4YrKr9B0pSySmHrBeme6HxTQgeS9V6t9J-oyioLT-146e2cFTO1Y6f4r1vfAjoliu0R9cBWL9_2O-x_3Lt0KtgHNYGrfNoorKN-yfhF8F8gMA</recordid><startdate>1973</startdate><enddate>1973</enddate><creator>Jacobson, Lester B.</creator><general>Elsevier Inc</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>1973</creationdate><title>Spontaneous ventricular parasystole initiating ventricular tachycardia</title><author>Jacobson, Lester B.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c360t-99e3a100a4c24e1ddb4d685245d55f9c16c810a7fb2ac48d1e148d2e897593ec3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1973</creationdate><topic>Aged</topic><topic>Arrhythmias, Cardiac</topic><topic>Electrocardiography</topic><topic>Heart Conduction System - physiopathology</topic><topic>Heart Failure - etiology</topic><topic>Heart Rate</topic><topic>Heart Ventricles - physiopathology</topic><topic>Humans</topic><topic>Male</topic><topic>Pacemaker, Artificial</topic><topic>Quinidine - therapeutic use</topic><topic>Tachycardia - etiology</topic><topic>Tachycardia - therapy</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Jacobson, Lester B.</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Journal of electrocardiology</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Jacobson, Lester B.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Spontaneous ventricular parasystole initiating ventricular tachycardia</atitle><jtitle>Journal of electrocardiology</jtitle><addtitle>J Electrocardiol</addtitle><date>1973</date><risdate>1973</risdate><volume>6</volume><issue>1</issue><spage>63</spage><epage>70</epage><pages>63-70</pages><issn>0022-0736</issn><eissn>1532-8430</eissn><abstract>Ventricular parasystolic rhythms are rarely hemodynamically significant. However, ventricular parasystolic beats, like fixed rate ventricular pacing stimuli, have the theoretical potential to initiate repetitive beating when they fall in the vulnerable period. In the case presented here, spontaneously occurring ventricular parasystolic beats were able to initiate hemodynamically significant ventricular tachycardias. The ventricular tachycardia, however, did not disturb the parasystolic rhythm whose beats continued to occur as expected and were able to terminate the tachycardia. Thus, ventricular tachycardia could be terminated by a ventricular parasystolic beat which originated in the same focus as the ventricular parasystolic beat which initiated the tachycardia.</abstract><cop>United States</cop><pub>Elsevier Inc</pub><pmid>4691404</pmid><doi>10.1016/S0022-0736(73)80026-3</doi><tpages>8</tpages></addata></record> |
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subjects | Aged Arrhythmias, Cardiac Electrocardiography Heart Conduction System - physiopathology Heart Failure - etiology Heart Rate Heart Ventricles - physiopathology Humans Male Pacemaker, Artificial Quinidine - therapeutic use Tachycardia - etiology Tachycardia - therapy |
title | Spontaneous ventricular parasystole initiating ventricular tachycardia |
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