Thioredoxin binding protein-2 mediates metabolic adaptation in response to lipopolysaccharide in vivo

OBJECTIVES:Endotoxin triggers a reorganization of the energy metabolic pathway, including the promotion of fatty acid utilization to adapt to a high energy demand during endotoxemia. However, the factors responsible for the metabolic adaptation and characteristic pathologies resulting from defective...

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Veröffentlicht in:	Critical care medicine 2010-12, Vol.38 (12), p.2345-2351
Hauptverfasser:	Oka, Shin-ichi, Liu, Wenrui, Yoshihara, Eiji, Ahsan, Md. Kaimul, Ramos, Dorys Adriana Lopez, Son, Aoi, Okuyama, Hiroaki, Zhang, Li, Masutani, Hiroshi, Nakamura, Hajime, Yodoi, Junji
Format:	Artikel
Sprache:	eng
Schlagworte:	Adaptation, Physiological Animals Blood Chemical Analysis Blotting, Western Carrier Proteins - genetics Carrier Proteins - metabolism Cytokines - metabolism Disease Models, Animal Endotoxemia - drug therapy Endotoxemia - metabolism Endotoxemia - mortality Endotoxemia - physiopathology Fatty Acids - metabolism Glucose - metabolism Immunohistochemistry Injections, Intraperitoneal Kaplan-Meier Estimate Lipopolysaccharides - pharmacology Mice Mice, Inbred C57BL Mice, Knockout Random Allocation Reverse Transcriptase Polymerase Chain Reaction Thioredoxins - genetics Thioredoxins - metabolism
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