Possible Mechanism of Hemostasis Effected by Electrocoagulation
Summary Forty acute gastric ulcers were created in 10 rabbits. The bleeding was controlled by monopolar electrocoagulation in 20 ulcers, the remaining ulcers were left untreated as controls. Spontaneous intravascular thrombosis was precluded by heparinization. A histological assessment in the acute...
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| Veröffentlicht in: | Endoscopy 1984-03, Vol.16 (2), p.43-46 |
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| container_title | Endoscopy |
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| creator | Wara, P. Berg, V. Jacobsen, N.O. Casalnuovo, C. Amdrup, E. |
| description | Summary
Forty acute gastric ulcers were created in 10 rabbits. The bleeding was controlled by monopolar electrocoagulation in 20 ulcers, the remaining ulcers were left untreated as controls. Spontaneous intravascular thrombosis was precluded by heparinization. A histological assessment in the acute stage revealed significant morphological alterations in electrocoagulated ulcers, with fibrin deposition, decreased tissue stainability and swelling of the vessel walls, indicating heat-induced damage including cells of the vessel walls. An intravascular occlusive fibrin thrombosis demonstrated in all coagulated ulcers is suggested to be the probable mechanism of hemostasis. |
| doi_str_mv | 10.1055/s-2007-1018530 |
| format | Article |
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Forty acute gastric ulcers were created in 10 rabbits. The bleeding was controlled by monopolar electrocoagulation in 20 ulcers, the remaining ulcers were left untreated as controls. Spontaneous intravascular thrombosis was precluded by heparinization. A histological assessment in the acute stage revealed significant morphological alterations in electrocoagulated ulcers, with fibrin deposition, decreased tissue stainability and swelling of the vessel walls, indicating heat-induced damage including cells of the vessel walls. An intravascular occlusive fibrin thrombosis demonstrated in all coagulated ulcers is suggested to be the probable mechanism of hemostasis.</description><identifier>ISSN: 0013-726X</identifier><identifier>EISSN: 1438-8812</identifier><identifier>DOI: 10.1055/s-2007-1018530</identifier><identifier>PMID: 6714174</identifier><identifier>CODEN: ENDCAM</identifier><language>eng</language><publisher>Stuttgart: Thieme</publisher><subject>Animals ; Biological and medical sciences ; Disease Models, Animal ; Electrocoagulation ; Endoscopy ; Gastroenterology. Liver. Pancreas. Abdomen ; Hemostasis ; Malformations ; Medical sciences ; Original Contributions ; Peptic Ulcer Hemorrhage - pathology ; Peptic Ulcer Hemorrhage - surgery ; Rabbits ; Stomach Ulcer - pathology ; Stomach Ulcer - surgery ; Stomach. Duodenum. Small intestine. Colon. Rectum. Anus</subject><ispartof>Endoscopy, 1984-03, Vol.16 (2), p.43-46</ispartof><rights>Georg Thieme Verlag KG Stuttgart · New York</rights><rights>1984 INIST-CNRS</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c357t-a441aa0a0bbd58993adb26bf54f4be9d7c6010cd706a754fa852ac69198773aa3</citedby></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://www.thieme-connect.de/products/ejournals/pdf/10.1055/s-2007-1018530.pdf$$EPDF$$P50$$Gthieme$$H</linktopdf><link.rule.ids>314,776,780,3004,3005,27901,27902,54534</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=9628421$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/6714174$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Wara, P.</creatorcontrib><creatorcontrib>Berg, V.</creatorcontrib><creatorcontrib>Jacobsen, N.O.</creatorcontrib><creatorcontrib>Casalnuovo, C.</creatorcontrib><creatorcontrib>Amdrup, E.</creatorcontrib><title>Possible Mechanism of Hemostasis Effected by Electrocoagulation</title><title>Endoscopy</title><addtitle>Endoscopy</addtitle><description>Summary
Forty acute gastric ulcers were created in 10 rabbits. The bleeding was controlled by monopolar electrocoagulation in 20 ulcers, the remaining ulcers were left untreated as controls. Spontaneous intravascular thrombosis was precluded by heparinization. A histological assessment in the acute stage revealed significant morphological alterations in electrocoagulated ulcers, with fibrin deposition, decreased tissue stainability and swelling of the vessel walls, indicating heat-induced damage including cells of the vessel walls. An intravascular occlusive fibrin thrombosis demonstrated in all coagulated ulcers is suggested to be the probable mechanism of hemostasis.</description><subject>Animals</subject><subject>Biological and medical sciences</subject><subject>Disease Models, Animal</subject><subject>Electrocoagulation</subject><subject>Endoscopy</subject><subject>Gastroenterology. Liver. Pancreas. Abdomen</subject><subject>Hemostasis</subject><subject>Malformations</subject><subject>Medical sciences</subject><subject>Original Contributions</subject><subject>Peptic Ulcer Hemorrhage - pathology</subject><subject>Peptic Ulcer Hemorrhage - surgery</subject><subject>Rabbits</subject><subject>Stomach Ulcer - pathology</subject><subject>Stomach Ulcer - surgery</subject><subject>Stomach. Duodenum. Small intestine. Colon. Rectum. Anus</subject><issn>0013-726X</issn><issn>1438-8812</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1984</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNp1kDFPwzAQRi0EKqWwsiFlQGwp58SxnQmhqlCkIhhAYrMujk1TJXGxk6H_nlSN2JjO53v-Tn6EXFOYU8iy-xAnACKmQGWWwgmZUpbKWEqanJIpAE1jkfCvc3IRwvbQAmQTMuGCMirYlDy8uxCqojbRq9EbbKvQRM5GK9O40GGoQrS01ujOlFGxj5b1cPROO_zua-wq116SM4t1MFdjnZHPp-XHYhWv355fFo_rWKeZ6GJkjCICQlGUmczzFMsi4YXNmGWFyUuhOVDQpQCOYrhEmSWoeU5zKUSKmM7I3TF3591Pb0KnmipoU9fYGtcHJSkkMmd8AOdHUPvhZ95YtfNVg36vKKiDMRXUwZgajQ0PbsbkvmhM-YePiob57TjHoLG2HltdhT8s54lkCR2w-Ih1m8o0Rm1d79vByH9rfwEpbICq</recordid><startdate>198403</startdate><enddate>198403</enddate><creator>Wara, P.</creator><creator>Berg, V.</creator><creator>Jacobsen, N.O.</creator><creator>Casalnuovo, C.</creator><creator>Amdrup, E.</creator><general>Thieme</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>198403</creationdate><title>Possible Mechanism of Hemostasis Effected by Electrocoagulation</title><author>Wara, P. ; Berg, V. ; Jacobsen, N.O. ; Casalnuovo, C. ; Amdrup, E.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c357t-a441aa0a0bbd58993adb26bf54f4be9d7c6010cd706a754fa852ac69198773aa3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1984</creationdate><topic>Animals</topic><topic>Biological and medical sciences</topic><topic>Disease Models, Animal</topic><topic>Electrocoagulation</topic><topic>Endoscopy</topic><topic>Gastroenterology. Liver. Pancreas. Abdomen</topic><topic>Hemostasis</topic><topic>Malformations</topic><topic>Medical sciences</topic><topic>Original Contributions</topic><topic>Peptic Ulcer Hemorrhage - pathology</topic><topic>Peptic Ulcer Hemorrhage - surgery</topic><topic>Rabbits</topic><topic>Stomach Ulcer - pathology</topic><topic>Stomach Ulcer - surgery</topic><topic>Stomach. Duodenum. Small intestine. Colon. Rectum. Anus</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Wara, P.</creatorcontrib><creatorcontrib>Berg, V.</creatorcontrib><creatorcontrib>Jacobsen, N.O.</creatorcontrib><creatorcontrib>Casalnuovo, C.</creatorcontrib><creatorcontrib>Amdrup, E.</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Endoscopy</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Wara, P.</au><au>Berg, V.</au><au>Jacobsen, N.O.</au><au>Casalnuovo, C.</au><au>Amdrup, E.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Possible Mechanism of Hemostasis Effected by Electrocoagulation</atitle><jtitle>Endoscopy</jtitle><addtitle>Endoscopy</addtitle><date>1984-03</date><risdate>1984</risdate><volume>16</volume><issue>2</issue><spage>43</spage><epage>46</epage><pages>43-46</pages><issn>0013-726X</issn><eissn>1438-8812</eissn><coden>ENDCAM</coden><abstract>Summary
Forty acute gastric ulcers were created in 10 rabbits. The bleeding was controlled by monopolar electrocoagulation in 20 ulcers, the remaining ulcers were left untreated as controls. Spontaneous intravascular thrombosis was precluded by heparinization. A histological assessment in the acute stage revealed significant morphological alterations in electrocoagulated ulcers, with fibrin deposition, decreased tissue stainability and swelling of the vessel walls, indicating heat-induced damage including cells of the vessel walls. An intravascular occlusive fibrin thrombosis demonstrated in all coagulated ulcers is suggested to be the probable mechanism of hemostasis.</abstract><cop>Stuttgart</cop><cop>New York, NY</cop><pub>Thieme</pub><pmid>6714174</pmid><doi>10.1055/s-2007-1018530</doi><tpages>4</tpages></addata></record> |
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| ispartof | Endoscopy, 1984-03, Vol.16 (2), p.43-46 |
| issn | 0013-726X 1438-8812 |
| language | eng |
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| source | MEDLINE; Thieme Connect Journals |
| subjects | Animals Biological and medical sciences Disease Models, Animal Electrocoagulation Endoscopy Gastroenterology. Liver. Pancreas. Abdomen Hemostasis Malformations Medical sciences Original Contributions Peptic Ulcer Hemorrhage - pathology Peptic Ulcer Hemorrhage - surgery Rabbits Stomach Ulcer - pathology Stomach Ulcer - surgery Stomach. Duodenum. Small intestine. Colon. Rectum. Anus |
| title | Possible Mechanism of Hemostasis Effected by Electrocoagulation |
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