Intestinal Lymphonodular Hyperplasia of Childhood: Patterns of Presentation
this retrospective analysis we searched for a constelation of signs or symptoms attributable to childhood mphonodular hyperplasia (LNH). Of 147 children with ocumented LNH reviewed, 43% had lesions in the small owel, and 57% in the large bowel. Children in this study presented with complaints of abd...
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Veröffentlicht in: | Journal of clinical gastroenterology 1991-04, Vol.13 (2), p.163-166 |
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description | this retrospective analysis we searched for a constelation of signs or symptoms attributable to childhood mphonodular hyperplasia (LNH). Of 147 children with ocumented LNH reviewed, 43% had lesions in the small owel, and 57% in the large bowel. Children in this study presented with complaints of abdominal pain (58%) and light red blood per rectum (32%). Physical examination evealed little except right lower quadrant (RLQ) abdommal tenderness and “fullness” in 35%. The pain was eriumbilical, dull-cramping, rarely acute, and nonradiating. The hematochezia was most commonly streaky red mucoid strands adhering to the stools, with no associated tenesmus. Three clinical patterns emerged(a) Unler 1 year of age most patients were male, with painless leeding and pancolonic LNH. (b) Between 2 and 6 years, though the LNH was predominantly colonic, pain and leeding occurred equally. (c) From 7 years old on, the ain symptom was abdominal pain, but LNH distribution has nearly equal between the small bowel and the colon. o date, our long-term follow-up of the children with olated LNH has revealed no sequelae. |
doi_str_mv | 10.1097/00004836-199104000-00009 |
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Of 147 children with ocumented LNH reviewed, 43% had lesions in the small owel, and 57% in the large bowel. Children in this study presented with complaints of abdominal pain (58%) and light red blood per rectum (32%). Physical examination evealed little except right lower quadrant (RLQ) abdommal tenderness and “fullness” in 35%. The pain was eriumbilical, dull-cramping, rarely acute, and nonradiating. The hematochezia was most commonly streaky red mucoid strands adhering to the stools, with no associated tenesmus. Three clinical patterns emerged(a) Unler 1 year of age most patients were male, with painless leeding and pancolonic LNH. (b) Between 2 and 6 years, though the LNH was predominantly colonic, pain and leeding occurred equally. (c) From 7 years old on, the ain symptom was abdominal pain, but LNH distribution has nearly equal between the small bowel and the colon. o date, our long-term follow-up of the children with olated LNH has revealed no sequelae.</description><identifier>ISSN: 0192-0790</identifier><identifier>EISSN: 1539-2031</identifier><identifier>DOI: 10.1097/00004836-199104000-00009</identifier><identifier>PMID: 2033223</identifier><identifier>CODEN: JCGADC</identifier><language>eng</language><publisher>Hagerstown, MD: Lippincott-Raven Publishers</publisher><subject>Abdominal Pain - etiology ; Biological and medical sciences ; Child ; Colon - pathology ; Colonic Diseases - complications ; Colonic Diseases - pathology ; Female ; Gastroenterology. Liver. Pancreas. Abdomen ; Gastrointestinal Hemorrhage - etiology ; Humans ; Hyperplasia ; Ileal Diseases - complications ; Ileal Diseases - pathology ; Ileum - pathology ; Lymphatic Diseases - complications ; Lymphatic Diseases - pathology ; Lymphoid Tissue - pathology ; Male ; Medical sciences ; Other diseases. Semiology ; Rectum ; Stomach. Duodenum. Small intestine. Colon. Rectum. Anus</subject><ispartof>Journal of clinical gastroenterology, 1991-04, Vol.13 (2), p.163-166</ispartof><rights>Lippincott-Raven Publishers.</rights><rights>1991 INIST-CNRS</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,780,784,27924,27925</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=19680331$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/2033223$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Colón, A R</creatorcontrib><creatorcontrib>DiPalma, J S</creatorcontrib><creatorcontrib>Leftridge, C A</creatorcontrib><title>Intestinal Lymphonodular Hyperplasia of Childhood: Patterns of Presentation</title><title>Journal of clinical gastroenterology</title><addtitle>J Clin Gastroenterol</addtitle><description>this retrospective analysis we searched for a constelation of signs or symptoms attributable to childhood mphonodular hyperplasia (LNH). Of 147 children with ocumented LNH reviewed, 43% had lesions in the small owel, and 57% in the large bowel. Children in this study presented with complaints of abdominal pain (58%) and light red blood per rectum (32%). Physical examination evealed little except right lower quadrant (RLQ) abdommal tenderness and “fullness” in 35%. The pain was eriumbilical, dull-cramping, rarely acute, and nonradiating. The hematochezia was most commonly streaky red mucoid strands adhering to the stools, with no associated tenesmus. Three clinical patterns emerged(a) Unler 1 year of age most patients were male, with painless leeding and pancolonic LNH. (b) Between 2 and 6 years, though the LNH was predominantly colonic, pain and leeding occurred equally. (c) From 7 years old on, the ain symptom was abdominal pain, but LNH distribution has nearly equal between the small bowel and the colon. o date, our long-term follow-up of the children with olated LNH has revealed no sequelae.</description><subject>Abdominal Pain - etiology</subject><subject>Biological and medical sciences</subject><subject>Child</subject><subject>Colon - pathology</subject><subject>Colonic Diseases - complications</subject><subject>Colonic Diseases - pathology</subject><subject>Female</subject><subject>Gastroenterology. Liver. Pancreas. Abdomen</subject><subject>Gastrointestinal Hemorrhage - etiology</subject><subject>Humans</subject><subject>Hyperplasia</subject><subject>Ileal Diseases - complications</subject><subject>Ileal Diseases - pathology</subject><subject>Ileum - pathology</subject><subject>Lymphatic Diseases - complications</subject><subject>Lymphatic Diseases - pathology</subject><subject>Lymphoid Tissue - pathology</subject><subject>Male</subject><subject>Medical sciences</subject><subject>Other diseases. Semiology</subject><subject>Rectum</subject><subject>Stomach. Duodenum. Small intestine. Colon. Rectum. Anus</subject><issn>0192-0790</issn><issn>1539-2031</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1991</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNp1kU1PxCAQhonR6PrxE0x60Vt1KAWKN7PxK26iBz2TkaVplS0V2mz238u6q57kMuF93xnIM4RkFC4oKHkJ6ZQVEzlVikKZbvlaUjtkQjlTeQGM7pIJUFXkIBUckMMY3wGoZIzuk_3ks6JgE_L40A02Dm2HLputFn3jOz8fHYbsftXb0DuMLWa-zqZN6-aN9_Or7BmHwYYuruXnYKPtBhxa3x2TvRpdtCfbekReb29epvf57OnuYXo9yw1joHIqsDSVlFwhR2UliDdRQlnXXMgKKyqlAFVxIwqOFCi3pTHKFoiS1RIB2BE538ztg_8c0-_1oo3GOoed9WPUFXBRCi5SsNoETfAxBlvrPrQLDCtNQa856h-O-pfjt6RS6-n2jfFtYee_jVtwyT_b-hgNujpgZ9r4N1-JKiVpypWb3NK7BC1-uHFpg24suqHR_62RfQFka4kq</recordid><startdate>199104</startdate><enddate>199104</enddate><creator>Colón, A R</creator><creator>DiPalma, J S</creator><creator>Leftridge, C A</creator><general>Lippincott-Raven Publishers</general><general>Lippincott Williams & Wilkins</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope><scope>8BM</scope></search><sort><creationdate>199104</creationdate><title>Intestinal Lymphonodular Hyperplasia of Childhood: Patterns of Presentation</title><author>Colón, A R ; DiPalma, J S ; Leftridge, C A</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c3309-16a4c87759a5a9e706b6404ff5678a817760985c625a1015e4cc9e2aa73f7a003</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1991</creationdate><topic>Abdominal Pain - etiology</topic><topic>Biological and medical sciences</topic><topic>Child</topic><topic>Colon - pathology</topic><topic>Colonic Diseases - complications</topic><topic>Colonic Diseases - pathology</topic><topic>Female</topic><topic>Gastroenterology. Liver. Pancreas. Abdomen</topic><topic>Gastrointestinal Hemorrhage - etiology</topic><topic>Humans</topic><topic>Hyperplasia</topic><topic>Ileal Diseases - complications</topic><topic>Ileal Diseases - pathology</topic><topic>Ileum - pathology</topic><topic>Lymphatic Diseases - complications</topic><topic>Lymphatic Diseases - pathology</topic><topic>Lymphoid Tissue - pathology</topic><topic>Male</topic><topic>Medical sciences</topic><topic>Other diseases. Semiology</topic><topic>Rectum</topic><topic>Stomach. Duodenum. Small intestine. Colon. Rectum. Anus</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Colón, A R</creatorcontrib><creatorcontrib>DiPalma, J S</creatorcontrib><creatorcontrib>Leftridge, C A</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><collection>ComDisDome</collection><jtitle>Journal of clinical gastroenterology</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Colón, A R</au><au>DiPalma, J S</au><au>Leftridge, C A</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Intestinal Lymphonodular Hyperplasia of Childhood: Patterns of Presentation</atitle><jtitle>Journal of clinical gastroenterology</jtitle><addtitle>J Clin Gastroenterol</addtitle><date>1991-04</date><risdate>1991</risdate><volume>13</volume><issue>2</issue><spage>163</spage><epage>166</epage><pages>163-166</pages><issn>0192-0790</issn><eissn>1539-2031</eissn><coden>JCGADC</coden><abstract>this retrospective analysis we searched for a constelation of signs or symptoms attributable to childhood mphonodular hyperplasia (LNH). Of 147 children with ocumented LNH reviewed, 43% had lesions in the small owel, and 57% in the large bowel. Children in this study presented with complaints of abdominal pain (58%) and light red blood per rectum (32%). Physical examination evealed little except right lower quadrant (RLQ) abdommal tenderness and “fullness” in 35%. The pain was eriumbilical, dull-cramping, rarely acute, and nonradiating. The hematochezia was most commonly streaky red mucoid strands adhering to the stools, with no associated tenesmus. Three clinical patterns emerged(a) Unler 1 year of age most patients were male, with painless leeding and pancolonic LNH. (b) Between 2 and 6 years, though the LNH was predominantly colonic, pain and leeding occurred equally. (c) From 7 years old on, the ain symptom was abdominal pain, but LNH distribution has nearly equal between the small bowel and the colon. o date, our long-term follow-up of the children with olated LNH has revealed no sequelae.</abstract><cop>Hagerstown, MD</cop><pub>Lippincott-Raven Publishers</pub><pmid>2033223</pmid><doi>10.1097/00004836-199104000-00009</doi><tpages>4</tpages></addata></record> |
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subjects | Abdominal Pain - etiology Biological and medical sciences Child Colon - pathology Colonic Diseases - complications Colonic Diseases - pathology Female Gastroenterology. Liver. Pancreas. Abdomen Gastrointestinal Hemorrhage - etiology Humans Hyperplasia Ileal Diseases - complications Ileal Diseases - pathology Ileum - pathology Lymphatic Diseases - complications Lymphatic Diseases - pathology Lymphoid Tissue - pathology Male Medical sciences Other diseases. Semiology Rectum Stomach. Duodenum. Small intestine. Colon. Rectum. Anus |
title | Intestinal Lymphonodular Hyperplasia of Childhood: Patterns of Presentation |
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