Nitroprusside in vitro inhibits platelet aggregation and intracellular calcium translocation. Effect of haemoglobin

The biologically final active compound of nitrovasodilators is now supposed to be nitric oxide (NO), a labile substance identical to EDRF. The effects of nitroprusside on platelet functions were studied in vitro. Platelet aggregation induced by several stimuli (ADP, collagen, arachidonic acid and PA...

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Veröffentlicht in:	Thrombosis research 1991-01, Vol.61 (2), p.113-122
Hauptverfasser:	Pasqui, A.L., Capecchi, P.L., Ceccatelli, L., Mazza, S., Gistri, A., Laghi Pasini, F., Di Perri, T.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenosine Diphosphate - pharmacology Arachidonic Acid Arachidonic Acids - pharmacology Biological and medical sciences Blood Platelets - drug effects Blood Platelets - physiology Blood. Blood coagulation. Reticuloendothelial system calcium Calcium - blood Collagen - pharmacology Hemoglobins - pharmacology Humans In Vitro Techniques Intracellular Fluid - drug effects Intracellular Fluid - metabolism Medical sciences nitroprusside Nitroprusside - pharmacology Pharmacology. Drug treatments Platelet Activating Factor - pharmacology Platelet Aggregation - drug effects Platelet Aggregation Inhibitors - pharmacology platelets
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