Left ventricular mass regression after aortic valve replacement measured by ultrafast computed tomography
Left ventricular mass and function were measured using ultrafast computed tomography, and were correlated with clinical status in 17 patients with aortic stenosis and/or insufficiency undergoing aortic valve replacement or balloon valvuloplasty. Wall mass was 159 ± 38 gm/m 2 initially, decreased 25%...
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| Veröffentlicht in: | The American heart journal 1990-10, Vol.120 (4), p.919-927 |
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| creator | Kurnik, Peter B. Innerfield, Michael Wachspress, Joseph D. Eldredge, W.Jay Waxman, Harvey L. |
| description | Left ventricular mass and function were measured using ultrafast computed tomography, and were correlated with clinical status in 17 patients with aortic stenosis and/or insufficiency undergoing aortic valve replacement or balloon valvuloplasty. Wall mass was 159 ± 38 gm/m
2 initially, decreased 25% to 116 ± 29 gm/m
2 at 4 month (
p < 0.001), and decreased a total of 34% to 105 ± 33 gm/m
2 at 8 months after valve repair. By 8 months not only was the mean wall mass within the normal range, but only three patients retained abnormal hypertrophy. Ejection fraction increased 8% (
p = 0.06). Clinical function improved in all patients, with only three patients remaining outside of New York Heart Association functional class 1 at 8 months. Regression of ventricular mass into the normal range correlated with attainment of class I functional status (
p < 0.02), despite a lack of increase of ejection fraction. The single patient followed for 8 months after valvuloplasty had minor wall mass regression and minor clinical improvement. |
| doi_str_mv | 10.1016/0002-8703(90)90211-F |
| format | Article |
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2 initially, decreased 25% to 116 ± 29 gm/m
2 at 4 month (
p < 0.001), and decreased a total of 34% to 105 ± 33 gm/m
2 at 8 months after valve repair. By 8 months not only was the mean wall mass within the normal range, but only three patients retained abnormal hypertrophy. Ejection fraction increased 8% (
p = 0.06). Clinical function improved in all patients, with only three patients remaining outside of New York Heart Association functional class 1 at 8 months. Regression of ventricular mass into the normal range correlated with attainment of class I functional status (
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2 initially, decreased 25% to 116 ± 29 gm/m
2 at 4 month (
p < 0.001), and decreased a total of 34% to 105 ± 33 gm/m
2 at 8 months after valve repair. By 8 months not only was the mean wall mass within the normal range, but only three patients retained abnormal hypertrophy. Ejection fraction increased 8% (
p = 0.06). Clinical function improved in all patients, with only three patients remaining outside of New York Heart Association functional class 1 at 8 months. Regression of ventricular mass into the normal range correlated with attainment of class I functional status (
p < 0.02), despite a lack of increase of ejection fraction. The single patient followed for 8 months after valvuloplasty had minor wall mass regression and minor clinical improvement.</description><subject>Aortic Valve Insufficiency - surgery</subject><subject>Aortic Valve Insufficiency - therapy</subject><subject>Aortic Valve Stenosis - surgery</subject><subject>Aortic Valve Stenosis - therapy</subject><subject>Biological and medical sciences</subject><subject>Cardiology. Vascular system</subject><subject>Cardiomegaly - diagnostic imaging</subject><subject>Cardiomegaly - physiopathology</subject><subject>Catheterization</subject><subject>Endocardial and cardiac valvular diseases</subject><subject>Follow-Up Studies</subject><subject>Heart</subject><subject>Heart Valve Prosthesis</subject><subject>Humans</subject><subject>Medical sciences</subject><subject>Tomography, X-Ray Computed - methods</subject><subject>Ventricular Function, Left - physiology</subject><issn>0002-8703</issn><issn>1097-6744</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1990</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNp9kE1r3DAQhkVpSTdp_0EKujS0Bzf68MrWJRBCNy0s9JKcxVgeJSq25Urywv77aLtLeutpmHk_GB5CLjn7xhlX14wxUbUNk180-6qZ4LzavCErznRTqaau35LVq-U9OU_pd1mVaNUZORO8XjeyXhG_RZfpDqccvV0GiHSElGjEp4gp-TBRcBkjhRCzt3QHww6LOg9gcSwpOiKkJWJPuz1dhhzBQcrUhnFecrnmMIanCPPz_gN552BI-PE0L8jj5vvD3Y9q--v-593ttrKybXKlW71miEL1oFVtuwY4h75FJVACaKEaQM6FZNhpKbXuZFs70SunwVlue3lBro69cwx_FkzZjD5ZHAaYMCzJtAVCLda8GOuj0caQUkRn5uhHiHvDmTkQNgd85oDPaGb-EjabEvt06l-6EfvX0Alp0T-fdEgWBhdhsj796y5ft2yti-_m6MMCY-cxmmQ9ThZ7H9Fm0wf__0deABr7meY</recordid><startdate>19901001</startdate><enddate>19901001</enddate><creator>Kurnik, Peter B.</creator><creator>Innerfield, Michael</creator><creator>Wachspress, Joseph D.</creator><creator>Eldredge, W.Jay</creator><creator>Waxman, Harvey L.</creator><general>Mosby, Inc</general><general>Elsevier</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>19901001</creationdate><title>Left ventricular mass regression after aortic valve replacement measured by ultrafast computed tomography</title><author>Kurnik, Peter B. ; Innerfield, Michael ; Wachspress, Joseph D. ; Eldredge, W.Jay ; Waxman, Harvey L.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c387t-98950ee26da964cb7a11ad8e62e3aa9267ae11230eb93399b384f2d6f9afc1cd3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1990</creationdate><topic>Aortic Valve Insufficiency - surgery</topic><topic>Aortic Valve Insufficiency - therapy</topic><topic>Aortic Valve Stenosis - surgery</topic><topic>Aortic Valve Stenosis - therapy</topic><topic>Biological and medical sciences</topic><topic>Cardiology. Vascular system</topic><topic>Cardiomegaly - diagnostic imaging</topic><topic>Cardiomegaly - physiopathology</topic><topic>Catheterization</topic><topic>Endocardial and cardiac valvular diseases</topic><topic>Follow-Up Studies</topic><topic>Heart</topic><topic>Heart Valve Prosthesis</topic><topic>Humans</topic><topic>Medical sciences</topic><topic>Tomography, X-Ray Computed - methods</topic><topic>Ventricular Function, Left - physiology</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Kurnik, Peter B.</creatorcontrib><creatorcontrib>Innerfield, Michael</creatorcontrib><creatorcontrib>Wachspress, Joseph D.</creatorcontrib><creatorcontrib>Eldredge, W.Jay</creatorcontrib><creatorcontrib>Waxman, Harvey L.</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>The American heart journal</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Kurnik, Peter B.</au><au>Innerfield, Michael</au><au>Wachspress, Joseph D.</au><au>Eldredge, W.Jay</au><au>Waxman, Harvey L.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Left ventricular mass regression after aortic valve replacement measured by ultrafast computed tomography</atitle><jtitle>The American heart journal</jtitle><addtitle>Am Heart J</addtitle><date>1990-10-01</date><risdate>1990</risdate><volume>120</volume><issue>4</issue><spage>919</spage><epage>927</epage><pages>919-927</pages><issn>0002-8703</issn><eissn>1097-6744</eissn><coden>AHJOA2</coden><abstract>Left ventricular mass and function were measured using ultrafast computed tomography, and were correlated with clinical status in 17 patients with aortic stenosis and/or insufficiency undergoing aortic valve replacement or balloon valvuloplasty. Wall mass was 159 ± 38 gm/m
2 initially, decreased 25% to 116 ± 29 gm/m
2 at 4 month (
p < 0.001), and decreased a total of 34% to 105 ± 33 gm/m
2 at 8 months after valve repair. By 8 months not only was the mean wall mass within the normal range, but only three patients retained abnormal hypertrophy. Ejection fraction increased 8% (
p = 0.06). Clinical function improved in all patients, with only three patients remaining outside of New York Heart Association functional class 1 at 8 months. Regression of ventricular mass into the normal range correlated with attainment of class I functional status (
p < 0.02), despite a lack of increase of ejection fraction. The single patient followed for 8 months after valvuloplasty had minor wall mass regression and minor clinical improvement.</abstract><cop>New York, NY</cop><pub>Mosby, Inc</pub><pmid>2145734</pmid><doi>10.1016/0002-8703(90)90211-F</doi><tpages>9</tpages></addata></record> |
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| language | eng |
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| source | MEDLINE; Elsevier ScienceDirect Journals Complete |
| subjects | Aortic Valve Insufficiency - surgery Aortic Valve Insufficiency - therapy Aortic Valve Stenosis - surgery Aortic Valve Stenosis - therapy Biological and medical sciences Cardiology. Vascular system Cardiomegaly - diagnostic imaging Cardiomegaly - physiopathology Catheterization Endocardial and cardiac valvular diseases Follow-Up Studies Heart Heart Valve Prosthesis Humans Medical sciences Tomography, X-Ray Computed - methods Ventricular Function, Left - physiology |
| title | Left ventricular mass regression after aortic valve replacement measured by ultrafast computed tomography |
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