p75-Deficient sensory axons are immunoreactive for the glycoprotein L1 in mice overexpressing nerve growth factor

Nerve growth factor (NGF) regulates the expression of the glycoprotein L1 among neural cell populations. The purpose of this investigation was to determine whether NGF equally affects the immunolocalization of L1 on both sympathetic and sensory axons, and whether the functional expression of the p75...

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Veröffentlicht in:	Brain research 1998-07, Vol.798 (1), p.184-194
Hauptverfasser:	Walsh, Gregory S, Petruccelli, Karen, Kawaja, Michael D
Format:	Artikel
Sprache:	eng
Schlagworte:	Adhesion molecule Animals Axons - metabolism Biological and medical sciences Calcitonin Gene-Related Peptide - metabolism Cerebellum - metabolism Fundamental and applied biological sciences. Psychology Glycoprotein Immunohistochemistry Leukocyte L1 Antigen Complex Membrane Glycoproteins - metabolism Mice Mice, Inbred C57BL Mice, Transgenic - genetics Mutation Nerve Growth Factors - genetics Nerve Growth Factors - metabolism Neural Cell Adhesion Molecules - metabolism Neurons, Afferent - metabolism Neurotrophin Peripheral nervous system. Autonomic nervous system. Neuromuscular transmission. Ganglionic transmission. Electric organ PNS Receptors, Nerve Growth Factor - deficiency Rhombencephalon - metabolism Tyrosine 3-Monooxygenase - metabolism Vertebrates: nervous system and sense organs
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