p53 and K-ras status in duodenal adenomas in familial adenomatous polyposis

Background The genetic alterations in patients with familial adenomatous polyposis (FAP) and duodenal adenomas are poorly characterized when compared with data relating to colorectal tumorigenesis in the same patients. Methods Point mutation of the K‐ras oncogene and point mutation and overexpressio...

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Veröffentlicht in:	British journal of surgery 1997-06, Vol.84 (6), p.826-829
Hauptverfasser:	Kashiwagi, H., Spigelman, A. D., Talbot, I. C., Debinski, H. S., McKie, A. B., Lemoine, N. R., Phillips, R. K. S.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenoma - genetics Adenoma - metabolism Adenomatous Polyposis Coli - genetics Adenomatous Polyposis Coli - metabolism Adult Aged Biological and medical sciences Biopsy Duodenal Neoplasms - genetics Duodenal Neoplasms - metabolism Exons - genetics Female Gastroenterology. Liver. Pancreas. Abdomen Genes, p53 - genetics Genes, ras - genetics Humans Immunohistochemistry Male Medical sciences Middle Aged Point Mutation Polymerase Chain Reaction Stomach. Duodenum. Small intestine. Colon. Rectum. Anus Tumors
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