Etiology of otospongiotic sensorineural losses
The etiology of otospongiotic-otosclerotic disease is enzymatic; the proteolytic enzymes released by the otospongiotic-otosclerotic foci damage the inner ear and are also the basis of the bony rebuilding of the OW niche leading to stapedial fixation. The trigger may be an autoimmune process due to t...
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Veröffentlicht in: | The American journal of otology (New York, N.Y.) N.Y.), 1989-03, Vol.10 (2), p.99-107 |
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creator | CAUSSE, J. R CAUSSE, J. B BRETLAU, P URIEL, J BERGES, J CHEVANCE, L. G SHAMBAUGH, G. E BASTIDE, J. M |
description | The etiology of otospongiotic-otosclerotic disease is enzymatic; the proteolytic enzymes released by the otospongiotic-otosclerotic foci damage the inner ear and are also the basis of the bony rebuilding of the OW niche leading to stapedial fixation. The trigger may be an autoimmune process due to the reaction of the enchondral otic capsule against the embryonic cartilaginous remnants, genetically determined to be located in the otic capsule and mainly in the fissula antefenestram. |
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The trigger may be an autoimmune process due to the reaction of the enchondral otic capsule against the embryonic cartilaginous remnants, genetically determined to be located in the otic capsule and mainly in the fissula antefenestram.</description><identifier>ISSN: 0192-9763</identifier><identifier>PMID: 2660582</identifier><identifier>CODEN: AJOTBN</identifier><language>eng</language><publisher>Hagerstown, MD: Lippincott</publisher><subject>Antibody Formation ; Autoantibodies - physiology ; Biological and medical sciences ; Ear, auditive nerve, cochleovestibular tract, facial nerve: diseases, semeiology ; Hearing Loss, Sensorineural - etiology ; Histiocytes - physiology ; Humans ; Medical sciences ; Non tumoral diseases ; Otorhinolaryngology. 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The trigger may be an autoimmune process due to the reaction of the enchondral otic capsule against the embryonic cartilaginous remnants, genetically determined to be located in the otic capsule and mainly in the fissula antefenestram.</description><subject>Antibody Formation</subject><subject>Autoantibodies - physiology</subject><subject>Biological and medical sciences</subject><subject>Ear, auditive nerve, cochleovestibular tract, facial nerve: diseases, semeiology</subject><subject>Hearing Loss, Sensorineural - etiology</subject><subject>Histiocytes - physiology</subject><subject>Humans</subject><subject>Medical sciences</subject><subject>Non tumoral diseases</subject><subject>Otorhinolaryngology. 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M</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Etiology of otospongiotic sensorineural losses</atitle><jtitle>The American journal of otology (New York, N.Y.)</jtitle><addtitle>Am J Otol</addtitle><date>1989-03-01</date><risdate>1989</risdate><volume>10</volume><issue>2</issue><spage>99</spage><epage>107</epage><pages>99-107</pages><issn>0192-9763</issn><coden>AJOTBN</coden><abstract>The etiology of otospongiotic-otosclerotic disease is enzymatic; the proteolytic enzymes released by the otospongiotic-otosclerotic foci damage the inner ear and are also the basis of the bony rebuilding of the OW niche leading to stapedial fixation. 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subjects | Antibody Formation Autoantibodies - physiology Biological and medical sciences Ear, auditive nerve, cochleovestibular tract, facial nerve: diseases, semeiology Hearing Loss, Sensorineural - etiology Histiocytes - physiology Humans Medical sciences Non tumoral diseases Otorhinolaryngology. Stomatology Otosclerosis - enzymology Otosclerosis - immunology Otosclerosis - physiopathology Peptide Hydrolases - physiology |
title | Etiology of otospongiotic sensorineural losses |
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