aex-3 Encodes a Novel Regulator of Presynaptic Activity in C. elegans

C. elegans aex-3mutations cause pleiotropic behavioral defects that are suggestive of reduced synaptic transmission. aex-3mutations also show strong genetic interactions with mutations in unc-31and unc-64, two other genes implicated in synaptic transmission. Physiological and pharmacological studies...

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Veröffentlicht in:	Neuron (Cambridge, Mass.) Mass.), 1997-04, Vol.18 (4), p.613-622
Hauptverfasser:	Iwasaki, Kouichi, Staunton, Jane, Saifee, Owais, Nonet, Michael, Thomas, James H
Format:	Artikel
Sprache:	eng
Schlagworte:	Aldicarb - pharmacology Amino Acid Sequence Animals Behavior, Animal - physiology Caenorhabditis elegans - genetics Cholinesterase Inhibitors - pharmacology Chromosome Mapping Electrophysiology Genes GTP-Binding Proteins - metabolism Molecular Sequence Data Mutation Nervous System Physiological Phenomena Pharynx - innervation Presynaptic Terminals - physiology rab3 GTP-Binding Proteins Synapses - physiology Synaptic Transmission Tissue Distribution
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