Oral l-arginine improves endothelium-dependent dilatation and reduces monocyte adhesion to endothelial cells in young men with coronary artery disease

l-Arginine is the physiological substrate for nitric oxide synthesis by the vascular endothelium. In hypercholesterolaemic rabbits, oral l-arginine reduces atheroma, improves endothelium-dependent dilatation and reduces monocyte/endothelial cell adhesion. The effect of oral l-arginine on endothelial...

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Veröffentlicht in:Atherosclerosis 1997-03, Vol.129 (2), p.261-269
Hauptverfasser: Adams, Mark R, McCredie, Robyn, Jessup, Wendy, Robinson, Jacqui, Sullivan, David, Celermajer, David S
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Sprache:eng
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