Dysregulation of arachidonic acid release and metabolism by atopic mononuclear cells

Summary We studied the ability of monocytes to metabolize [3H]arachidonic acid (AA) provided exogenously by activated T cells, and the extent to which dexamethasone suppressed eicosanoid production by normal and atopic cells.[3H]AA metabolites were identified using a reverse‐phase high pressure liqu...

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Veröffentlicht in:	Clinical and experimental allergy 1989-01, Vol.19 (1), p.37-44
Hauptverfasser:	LANS, D.M., ROCKLIN, R.E.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Arachidonic Acid Arachidonic Acids - metabolism Arachidonic Acids - pharmacokinetics Asthma - pathology Biological and medical sciences Chromatography, High Pressure Liquid Dermatitis, Atopic - pathology Dexamethasone - pharmacology Eicosanoic Acids - metabolism Female General aspects Humans Hypersensitivity, Immediate - metabolism Hypersensitivity, Immediate - pathology Immunopathology Male Medical sciences Middle Aged Monocytes - metabolism Rhinitis, Allergic, Perennial - pathology T-Lymphocytes - metabolism Tritium
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