Microfibrosis Produces Electrical Load Variations Due to Loss of Side-to-Side Cell Connections; A Major Mechanism of Structural Heart Disease Arrhythmias

The purpose of this article is to demonstrate how adaptive changes in myocardial microstructure provide mechanisms for emergent new conduction disturbances that initiate reentrant arrhythmias. The mechanisms are based on discontinuous conduction phenomena produced by increases in cellular hading; th...

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Veröffentlicht in:	Pacing and clinical electrophysiology 1997-02, Vol.20 (2), p.397-413
Hauptverfasser:	SPACH, MADISON S., BOINEAU, JOHN P.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals arrhythmias Arrhythmias, Cardiac - etiology Arrhythmias, Cardiac - pathology Arrhythmias, Cardiac - physiopathology Atrial Function - physiology Coronary Disease - pathology Coronary Disease - physiopathology Electrophysiology Fibrosis Gap Junctions - metabolism Gap Junctions - physiology Heart Conduction System - physiology Heart Conduction System - physiopathology Humans Intercellular Junctions - metabolism Intercellular Junctions - physiology microfibrosis myocardial microstructure Myocardium - metabolism Myocardium - pathology structural heart disease
Online-Zugang:	Volltext
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