Cyclosporin A and verapamil enhancement of daunorubicin-produced nucleolar protein B23 translocation in daunorubicin-resistant and -sensitive human and murine tumor cells

It has recently been shown that anthracycline antibiotic-resistant tumor cells are less responsive to daunorubicin-stimulated B23 nucleolar phosphoprotein translocation than drug-sensitive cells. Since cyclosporin A and verapamil reverse primary acquired and secondary cross-resistance to daunorubici...

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Veröffentlicht in:	Cancer research (Chicago, Ill.) Ill.), 1989-02, Vol.49 (3), p.677-680
Hauptverfasser:	SWEET, P, CHAN, P. K, SLATER, L. M
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Antineoplastic agents B23 protein Biological and medical sciences Cell Line cyclosporin A Cyclosporins - pharmacology Daunorubicin - pharmacology Drug Resistance enhancement General aspects Humans induction Liver Neoplasms, Experimental - metabolism man Medical sciences Mice Nuclear Proteins - metabolism Pharmacology. Drug treatments Precursor Cell Lymphoblastic Leukemia-Lymphoma - metabolism translocation tumours verapamil Verapamil - pharmacology
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