Long-chain fatty acids suppress the induction of urea cycle enzyme genes by glucocorticoid action

In order to test the possibility that free fatty acids are the mediator of the abnormal expression of urea cycle enzyme genes in carnitine-deficient juvenile visceral steatosis (JVS) mice, the effects of fatty acids on urea cycle enzyme, carbamoylphosphate synthetase (CPS) and argininosuccinate synt...

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Veröffentlicht in:	FEBS letters 1996-12, Vol.399 (3), p.310-312
Hauptverfasser:	Tomomura, Mineko, Tomomura, Akito, Abdullah Abu Musa, Dewan Md, Saheki, Takeyori
Format:	Artikel
Sprache:	eng
Schlagworte:	12-O-tetradecanoylphorbol 13-acetate activator protein 1 Animals AP-1 Argininosuccinate Synthase - genetics Argininosuccinate synthetase ASS Carbamoyl-Phosphate Synthase (Ammonia) - genetics Carbamoylphosphate synthetase Carnitine Cells, Cultured CPS Dexamethasone - pharmacology Fatty acid Fatty Acids - pharmacology GAPDH Gene Expression Regulation, Enzymologic - drug effects Glucocorticoid glyceraldehyde-3-phosphate dehydrogenase Liver - cytology Liver - drug effects Liver - enzymology Male PEPCK phosphoenolpyruvate carboxykinase Rat cultured hepatocyte Rats Rats, Wistar RNA, Messenger - genetics TPA
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