Atrioventricular Conduction in Sick Sinus Syndrome
With the increasing recognition of the pacemaker syndrome and with the availability of newer methods of pacing therapy, the status of the atrioventricular (AV) conduction in patients with Sick Sinus Sydrome (SSS) becomes crucial in the choice of mode of pacing. At the Philippine Heart Center, from A...
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Veröffentlicht in: | Pacing and clinical electrophysiology 1988-11, Vol.11 (11), p.1636-1640 |
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description | With the increasing recognition of the pacemaker syndrome and with the availability of newer methods of pacing therapy, the status of the atrioventricular (AV) conduction in patients with Sick Sinus Sydrome (SSS) becomes crucial in the choice of mode of pacing. At the Philippine Heart Center, from April 1983 to November 1986, the sinus and AV node function studies of 46 patients who, by electrophysiological studies had sinus node dysfunction (SND)‐SN recovery time (SNRT) >1400 msec, were reviewed. These were arbitrarily classified according to duration of SNRT into: Group A—borderline SND, SNRT from 1,401 to 1,499 msec (n = 4); Group B—mild SND, SNRT from 1,500 to 2,499 msec fn = 25); Group G—moderate SND, SNRT from 2,500 to 3499 msec fn = 6); Group D— severe SND, SNRT of 3,500 m and above (n = 11). Out of the 46, 14 (30%) had concomittant AVN dysfunction (AVND)—antegrade block rate < 130 beats per minute (BPM). The percentage occurrence of AVND was noted as follows: Group A—25% (1/4); Group B—28% (7/25); Group G—50% (3/6); Group D—27% (3/11). Out of the 14 patients with concomittant AVND, 5(35%) had antegrade block rate < 100 BPM, 3 in Group R and 2 in Group D. The study shows that AVND occurs in only 30% of SSS patients. Its occurrence and severity has no bearing on the degree of SND. In these, antegrade block was at a rate higher than 100 BPM in the majority of patients. The data suggest that most of SSS patients may benefit from atrial demand pacemaker which therapeutically is of more advantage over the ventricular demand type inasmuch as AV synchrony is retained, thus pacemaker syndrome is prevented. |
doi_str_mv | 10.1111/j.1540-8159.1988.tb06287.x |
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BELEN O. ; MANALO, JUANCHO M. ; CHUA, WILLIAM T.</creator><creatorcontrib>CARISMA, MA. BELEN O. ; MANALO, JUANCHO M. ; CHUA, WILLIAM T.</creatorcontrib><description>With the increasing recognition of the pacemaker syndrome and with the availability of newer methods of pacing therapy, the status of the atrioventricular (AV) conduction in patients with Sick Sinus Sydrome (SSS) becomes crucial in the choice of mode of pacing. At the Philippine Heart Center, from April 1983 to November 1986, the sinus and AV node function studies of 46 patients who, by electrophysiological studies had sinus node dysfunction (SND)‐SN recovery time (SNRT) >1400 msec, were reviewed. These were arbitrarily classified according to duration of SNRT into: Group A—borderline SND, SNRT from 1,401 to 1,499 msec (n = 4); Group B—mild SND, SNRT from 1,500 to 2,499 msec fn = 25); Group G—moderate SND, SNRT from 2,500 to 3499 msec fn = 6); Group D— severe SND, SNRT of 3,500 m and above (n = 11). Out of the 46, 14 (30%) had concomittant AVN dysfunction (AVND)—antegrade block rate < 130 beats per minute (BPM). The percentage occurrence of AVND was noted as follows: Group A—25% (1/4); Group B—28% (7/25); Group G—50% (3/6); Group D—27% (3/11). Out of the 14 patients with concomittant AVND, 5(35%) had antegrade block rate < 100 BPM, 3 in Group R and 2 in Group D. The study shows that AVND occurs in only 30% of SSS patients. Its occurrence and severity has no bearing on the degree of SND. In these, antegrade block was at a rate higher than 100 BPM in the majority of patients. The data suggest that most of SSS patients may benefit from atrial demand pacemaker which therapeutically is of more advantage over the ventricular demand type inasmuch as AV synchrony is retained, thus pacemaker syndrome is prevented.</description><identifier>ISSN: 0147-8389</identifier><identifier>EISSN: 1540-8159</identifier><identifier>DOI: 10.1111/j.1540-8159.1988.tb06287.x</identifier><identifier>PMID: 2463525</identifier><language>eng</language><publisher>Oxford, UK: Blackwell Publishing Ltd</publisher><subject>"pacemaker syndrome" ; Adult ; Aged ; atrial demand pacemaker ; Atrioventricular Node - physiopathology ; AV node dysfunction ; Cardiac Pacing, Artificial ; Electrocardiography ; Female ; Heart Conduction System - physiopathology ; Humans ; Male ; Middle Aged ; sick sinus syndrome ; Sick Sinus Syndrome - physiopathology ; Sick Sinus Syndrome - therapy</subject><ispartof>Pacing and clinical electrophysiology, 1988-11, Vol.11 (11), p.1636-1640</ispartof><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c4096-41b6e731ceb1c2736aa7e397533e7e338072ac63e9177c420c0785920bb6d7593</citedby><cites>FETCH-LOGICAL-c4096-41b6e731ceb1c2736aa7e397533e7e338072ac63e9177c420c0785920bb6d7593</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://onlinelibrary.wiley.com/doi/pdf/10.1111%2Fj.1540-8159.1988.tb06287.x$$EPDF$$P50$$Gwiley$$H</linktopdf><linktohtml>$$Uhttps://onlinelibrary.wiley.com/doi/full/10.1111%2Fj.1540-8159.1988.tb06287.x$$EHTML$$P50$$Gwiley$$H</linktohtml><link.rule.ids>314,776,780,1411,27903,27904,45553,45554</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/2463525$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>CARISMA, MA. BELEN O.</creatorcontrib><creatorcontrib>MANALO, JUANCHO M.</creatorcontrib><creatorcontrib>CHUA, WILLIAM T.</creatorcontrib><title>Atrioventricular Conduction in Sick Sinus Syndrome</title><title>Pacing and clinical electrophysiology</title><addtitle>Pacing Clin Electrophysiol</addtitle><description>With the increasing recognition of the pacemaker syndrome and with the availability of newer methods of pacing therapy, the status of the atrioventricular (AV) conduction in patients with Sick Sinus Sydrome (SSS) becomes crucial in the choice of mode of pacing. At the Philippine Heart Center, from April 1983 to November 1986, the sinus and AV node function studies of 46 patients who, by electrophysiological studies had sinus node dysfunction (SND)‐SN recovery time (SNRT) >1400 msec, were reviewed. These were arbitrarily classified according to duration of SNRT into: Group A—borderline SND, SNRT from 1,401 to 1,499 msec (n = 4); Group B—mild SND, SNRT from 1,500 to 2,499 msec fn = 25); Group G—moderate SND, SNRT from 2,500 to 3499 msec fn = 6); Group D— severe SND, SNRT of 3,500 m and above (n = 11). Out of the 46, 14 (30%) had concomittant AVN dysfunction (AVND)—antegrade block rate < 130 beats per minute (BPM). The percentage occurrence of AVND was noted as follows: Group A—25% (1/4); Group B—28% (7/25); Group G—50% (3/6); Group D—27% (3/11). Out of the 14 patients with concomittant AVND, 5(35%) had antegrade block rate < 100 BPM, 3 in Group R and 2 in Group D. The study shows that AVND occurs in only 30% of SSS patients. Its occurrence and severity has no bearing on the degree of SND. In these, antegrade block was at a rate higher than 100 BPM in the majority of patients. The data suggest that most of SSS patients may benefit from atrial demand pacemaker which therapeutically is of more advantage over the ventricular demand type inasmuch as AV synchrony is retained, thus pacemaker syndrome is prevented.</description><subject>"pacemaker syndrome"</subject><subject>Adult</subject><subject>Aged</subject><subject>atrial demand pacemaker</subject><subject>Atrioventricular Node - physiopathology</subject><subject>AV node dysfunction</subject><subject>Cardiac Pacing, Artificial</subject><subject>Electrocardiography</subject><subject>Female</subject><subject>Heart Conduction System - physiopathology</subject><subject>Humans</subject><subject>Male</subject><subject>Middle Aged</subject><subject>sick sinus syndrome</subject><subject>Sick Sinus Syndrome - physiopathology</subject><subject>Sick Sinus Syndrome - therapy</subject><issn>0147-8389</issn><issn>1540-8159</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1988</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqVkF1LwzAUhoMoc05_glC88K41aZovr5xlTnHMwRQvQ5tmkK0fmrS6_XtbWnbvuTgn8J7zBB4AbhAMUFt32wCRCPocEREgwXlQp5CGnAX7EzA-RqdgDFHEfI65OAcXzm0hhBRGZARGYUQxCckYhNPamupHl-1QTZ5YL67KrFG1qUrPlN7aqF3bysZ560OZ2arQl-Bsk-ROXw1zAj6eZu_xs794m7_E04WvIiioH6GUaoaR0ilSIcM0SZjGghGMdfvAHLIwURRrgRhTUQgVZJyIEKYpzRgReAJue-6Xrb4b7WpZGKd0nielrhonGacQCYHbxft-UdnKOas38suaIrEHiaDshMmt7KzIzorshMlBmNy3x9fDL01a6Ox4Ohhq84c-_zW5PvyDLFfTeIYopi3C7xHG1Xp_RCR2JynDjMjP5VyS-PWRx2QpV_gPzqqJAQ</recordid><startdate>198811</startdate><enddate>198811</enddate><creator>CARISMA, MA. BELEN O.</creator><creator>MANALO, JUANCHO M.</creator><creator>CHUA, WILLIAM T.</creator><general>Blackwell Publishing Ltd</general><scope>BSCLL</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>198811</creationdate><title>Atrioventricular Conduction in Sick Sinus Syndrome</title><author>CARISMA, MA. BELEN O. ; MANALO, JUANCHO M. ; CHUA, WILLIAM T.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c4096-41b6e731ceb1c2736aa7e397533e7e338072ac63e9177c420c0785920bb6d7593</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1988</creationdate><topic>"pacemaker syndrome"</topic><topic>Adult</topic><topic>Aged</topic><topic>atrial demand pacemaker</topic><topic>Atrioventricular Node - physiopathology</topic><topic>AV node dysfunction</topic><topic>Cardiac Pacing, Artificial</topic><topic>Electrocardiography</topic><topic>Female</topic><topic>Heart Conduction System - physiopathology</topic><topic>Humans</topic><topic>Male</topic><topic>Middle Aged</topic><topic>sick sinus syndrome</topic><topic>Sick Sinus Syndrome - physiopathology</topic><topic>Sick Sinus Syndrome - therapy</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>CARISMA, MA. BELEN O.</creatorcontrib><creatorcontrib>MANALO, JUANCHO M.</creatorcontrib><creatorcontrib>CHUA, WILLIAM T.</creatorcontrib><collection>Istex</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Pacing and clinical electrophysiology</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>CARISMA, MA. BELEN O.</au><au>MANALO, JUANCHO M.</au><au>CHUA, WILLIAM T.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Atrioventricular Conduction in Sick Sinus Syndrome</atitle><jtitle>Pacing and clinical electrophysiology</jtitle><addtitle>Pacing Clin Electrophysiol</addtitle><date>1988-11</date><risdate>1988</risdate><volume>11</volume><issue>11</issue><spage>1636</spage><epage>1640</epage><pages>1636-1640</pages><issn>0147-8389</issn><eissn>1540-8159</eissn><abstract>With the increasing recognition of the pacemaker syndrome and with the availability of newer methods of pacing therapy, the status of the atrioventricular (AV) conduction in patients with Sick Sinus Sydrome (SSS) becomes crucial in the choice of mode of pacing. At the Philippine Heart Center, from April 1983 to November 1986, the sinus and AV node function studies of 46 patients who, by electrophysiological studies had sinus node dysfunction (SND)‐SN recovery time (SNRT) >1400 msec, were reviewed. These were arbitrarily classified according to duration of SNRT into: Group A—borderline SND, SNRT from 1,401 to 1,499 msec (n = 4); Group B—mild SND, SNRT from 1,500 to 2,499 msec fn = 25); Group G—moderate SND, SNRT from 2,500 to 3499 msec fn = 6); Group D— severe SND, SNRT of 3,500 m and above (n = 11). Out of the 46, 14 (30%) had concomittant AVN dysfunction (AVND)—antegrade block rate < 130 beats per minute (BPM). The percentage occurrence of AVND was noted as follows: Group A—25% (1/4); Group B—28% (7/25); Group G—50% (3/6); Group D—27% (3/11). Out of the 14 patients with concomittant AVND, 5(35%) had antegrade block rate < 100 BPM, 3 in Group R and 2 in Group D. The study shows that AVND occurs in only 30% of SSS patients. Its occurrence and severity has no bearing on the degree of SND. In these, antegrade block was at a rate higher than 100 BPM in the majority of patients. The data suggest that most of SSS patients may benefit from atrial demand pacemaker which therapeutically is of more advantage over the ventricular demand type inasmuch as AV synchrony is retained, thus pacemaker syndrome is prevented.</abstract><cop>Oxford, UK</cop><pub>Blackwell Publishing Ltd</pub><pmid>2463525</pmid><doi>10.1111/j.1540-8159.1988.tb06287.x</doi><tpages>5</tpages></addata></record> |
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subjects | "pacemaker syndrome" Adult Aged atrial demand pacemaker Atrioventricular Node - physiopathology AV node dysfunction Cardiac Pacing, Artificial Electrocardiography Female Heart Conduction System - physiopathology Humans Male Middle Aged sick sinus syndrome Sick Sinus Syndrome - physiopathology Sick Sinus Syndrome - therapy |
title | Atrioventricular Conduction in Sick Sinus Syndrome |
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