Heat Stress Improves Functional Recovery and Induces Synthesis of 27- and 70-kDa Heat Shock Proteins Without Preserving Sarcoplasmic Reticulum Function in the Ischemic Rat Heart

Heat stress (HS) and the subsequent expression of heat shock proteins has been shown to enhance post-ischemic functional recovery and reduce infarct size. The purpose of these experiments was to determine if HS pre-treatment preserves sarcoplasmic reticulum (SR) function, a cellular organelle that p...

Ausführliche Beschreibung