Stimulation of the hyaluronic acid levels of human synovial fibroblasts by recombinant human tumor necrosis factor α, tumor necrosis factor β (lymphotoxin), interleukin‐1α, and interleukin‐1β

Monocyte/macrophage polypeptides (monokines) alter the properties of synovial cells. This interaction could explain some of the properties of the inflamed synovium in rheumatic disease. Only recently has it been possible to test the action of purified monokines on the target synovial cells. We repor...

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Veröffentlicht in:	Arthritis and rheumatism 1988-10, Vol.31 (10), p.1281-1289
Hauptverfasser:	Butler, Debra M., Vitti, Gerard F., Leizer, Tali, Hamilton, John A.
Format:	Artikel
Sprache:	eng
Schlagworte:	Dexamethasone - pharmacology Fibroblasts - metabolism Humans Hyaluronic Acid - metabolism Interleukin-1 - pharmacology Interleukin-1beta Peptide Fragments - pharmacology Plasminogen Activators - metabolism Prostaglandin-Endoperoxide Synthases - metabolism Recombinant Proteins Stimulation, Chemical Synovial Membrane - cytology Synovial Membrane - metabolism Tretinoin - pharmacology Tumor Necrosis Factor-alpha - pharmacology
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