Spasticity and contracture: physiologic aspects of formation
Disruption of the upper motor neuron inhibitory pathways by stroke, brain trauma, or spinal cord injury leads to muscle spasticity. Spasticity is characterized by increased muscle tone, hyperactive reflexes, and possible clonus or rigidity. The increased muscle tone may result in loss of joint motio...
Gespeichert in:
Veröffentlicht in: | Clinical orthopaedics and related research 1988-08, Vol.233 (233), p.7-18 |
---|---|
Hauptverfasser: | , , |
Format: | Artikel |
Sprache: | eng |
Schlagworte: | |
Online-Zugang: | Volltext |
Tags: |
Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
|
container_end_page | 18 |
---|---|
container_issue | 233 |
container_start_page | 7 |
container_title | Clinical orthopaedics and related research |
container_volume | 233 |
creator | BOTTE, M. J NICKEL, V. L AKESON, W. H |
description | Disruption of the upper motor neuron inhibitory pathways by stroke, brain trauma, or spinal cord injury leads to muscle spasticity. Spasticity is characterized by increased muscle tone, hyperactive reflexes, and possible clonus or rigidity. The increased muscle tone may result in loss of joint motion, leading to contractures. Treatment of established contractures is difficult. Prevention of contractures by joint mobilization is emphasized as a goal in the management of patients with spasticity. |
doi_str_mv | 10.1097/00003086-198808000-00003 |
format | Article |
fullrecord | <record><control><sourceid>proquest_cross</sourceid><recordid>TN_cdi_proquest_miscellaneous_78355335</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><sourcerecordid>78355335</sourcerecordid><originalsourceid>FETCH-LOGICAL-c405t-b5759469913f9a90796ef1eb8b90cee02e4f6a12c73734ef5b854d8374d0ec393</originalsourceid><addsrcrecordid>eNo9kE1LxDAQhoMo67r6E4QexFs1n00iXmTxCxY8qOCtpGmikbapSXrov7fr1p3LMO_7zgw8AGQIXiEo-TWcikBR5EgKAcU05X_SAVgihkWOEMGHYDlJMpcYfRyDkxi_twnK8AIsCKQYE74Et6-9islpl8ZMdXWmfZeC0mkI5ibrv8bofOM_nc5U7I1OMfM2sz60KjnfnYIjq5pozua-Au8P92_rp3zz8vi8vtvkmkKW8opxJmkhJSJWKgm5LIxFphKVhNoYiA21hUJYc8IJNZZVgtFaEE5raDSRZAUud3f74H8GE1PZuqhN06jO-CGWXBDGCGFTUOyCOvgYg7FlH1yrwlgiWG7Blf_gyj24nTStns8_hqo19X5xJjX5F7OvolaNDarTLu5jHHJOOSO_qDV1BQ</addsrcrecordid><sourcetype>Aggregation Database</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>78355335</pqid></control><display><type>article</type><title>Spasticity and contracture: physiologic aspects of formation</title><source>MEDLINE</source><source>Journals@Ovid Complete</source><creator>BOTTE, M. J ; NICKEL, V. L ; AKESON, W. H</creator><creatorcontrib>BOTTE, M. J ; NICKEL, V. L ; AKESON, W. H</creatorcontrib><description>Disruption of the upper motor neuron inhibitory pathways by stroke, brain trauma, or spinal cord injury leads to muscle spasticity. Spasticity is characterized by increased muscle tone, hyperactive reflexes, and possible clonus or rigidity. The increased muscle tone may result in loss of joint motion, leading to contractures. Treatment of established contractures is difficult. Prevention of contractures by joint mobilization is emphasized as a goal in the management of patients with spasticity.</description><identifier>ISSN: 0009-921X</identifier><identifier>EISSN: 1528-1132</identifier><identifier>DOI: 10.1097/00003086-198808000-00003</identifier><identifier>PMID: 3042237</identifier><identifier>CODEN: CORTBR</identifier><language>eng</language><publisher>Heidelberg: Springer</publisher><subject>Animals ; Biological and medical sciences ; Contracture - physiopathology ; Contracture - prevention & control ; Contracture - therapy ; Humans ; Medical sciences ; Motor Neurons - physiology ; Muscle Spasticity - physiopathology ; Muscle Spasticity - prevention & control ; Muscle Spasticity - therapy ; Muscles - innervation ; Nervous system (semeiology, syndromes) ; Nervous system as a whole ; Neural Pathways - anatomy & histology ; Neural Pathways - physiology ; Neurology ; Neurons, Efferent - physiology</subject><ispartof>Clinical orthopaedics and related research, 1988-08, Vol.233 (233), p.7-18</ispartof><rights>1989 INIST-CNRS</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c405t-b5759469913f9a90796ef1eb8b90cee02e4f6a12c73734ef5b854d8374d0ec393</citedby></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>315,781,785,27929,27930</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=7077475$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/3042237$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>BOTTE, M. J</creatorcontrib><creatorcontrib>NICKEL, V. L</creatorcontrib><creatorcontrib>AKESON, W. H</creatorcontrib><title>Spasticity and contracture: physiologic aspects of formation</title><title>Clinical orthopaedics and related research</title><addtitle>Clin Orthop Relat Res</addtitle><description>Disruption of the upper motor neuron inhibitory pathways by stroke, brain trauma, or spinal cord injury leads to muscle spasticity. Spasticity is characterized by increased muscle tone, hyperactive reflexes, and possible clonus or rigidity. The increased muscle tone may result in loss of joint motion, leading to contractures. Treatment of established contractures is difficult. Prevention of contractures by joint mobilization is emphasized as a goal in the management of patients with spasticity.</description><subject>Animals</subject><subject>Biological and medical sciences</subject><subject>Contracture - physiopathology</subject><subject>Contracture - prevention & control</subject><subject>Contracture - therapy</subject><subject>Humans</subject><subject>Medical sciences</subject><subject>Motor Neurons - physiology</subject><subject>Muscle Spasticity - physiopathology</subject><subject>Muscle Spasticity - prevention & control</subject><subject>Muscle Spasticity - therapy</subject><subject>Muscles - innervation</subject><subject>Nervous system (semeiology, syndromes)</subject><subject>Nervous system as a whole</subject><subject>Neural Pathways - anatomy & histology</subject><subject>Neural Pathways - physiology</subject><subject>Neurology</subject><subject>Neurons, Efferent - physiology</subject><issn>0009-921X</issn><issn>1528-1132</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1988</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNo9kE1LxDAQhoMo67r6E4QexFs1n00iXmTxCxY8qOCtpGmikbapSXrov7fr1p3LMO_7zgw8AGQIXiEo-TWcikBR5EgKAcU05X_SAVgihkWOEMGHYDlJMpcYfRyDkxi_twnK8AIsCKQYE74Et6-9islpl8ZMdXWmfZeC0mkI5ibrv8bofOM_nc5U7I1OMfM2sz60KjnfnYIjq5pozua-Au8P92_rp3zz8vi8vtvkmkKW8opxJmkhJSJWKgm5LIxFphKVhNoYiA21hUJYc8IJNZZVgtFaEE5raDSRZAUud3f74H8GE1PZuqhN06jO-CGWXBDGCGFTUOyCOvgYg7FlH1yrwlgiWG7Blf_gyj24nTStns8_hqo19X5xJjX5F7OvolaNDarTLu5jHHJOOSO_qDV1BQ</recordid><startdate>19880801</startdate><enddate>19880801</enddate><creator>BOTTE, M. J</creator><creator>NICKEL, V. L</creator><creator>AKESON, W. H</creator><general>Springer</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>19880801</creationdate><title>Spasticity and contracture: physiologic aspects of formation</title><author>BOTTE, M. J ; NICKEL, V. L ; AKESON, W. H</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c405t-b5759469913f9a90796ef1eb8b90cee02e4f6a12c73734ef5b854d8374d0ec393</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1988</creationdate><topic>Animals</topic><topic>Biological and medical sciences</topic><topic>Contracture - physiopathology</topic><topic>Contracture - prevention & control</topic><topic>Contracture - therapy</topic><topic>Humans</topic><topic>Medical sciences</topic><topic>Motor Neurons - physiology</topic><topic>Muscle Spasticity - physiopathology</topic><topic>Muscle Spasticity - prevention & control</topic><topic>Muscle Spasticity - therapy</topic><topic>Muscles - innervation</topic><topic>Nervous system (semeiology, syndromes)</topic><topic>Nervous system as a whole</topic><topic>Neural Pathways - anatomy & histology</topic><topic>Neural Pathways - physiology</topic><topic>Neurology</topic><topic>Neurons, Efferent - physiology</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>BOTTE, M. J</creatorcontrib><creatorcontrib>NICKEL, V. L</creatorcontrib><creatorcontrib>AKESON, W. H</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Clinical orthopaedics and related research</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>BOTTE, M. J</au><au>NICKEL, V. L</au><au>AKESON, W. H</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Spasticity and contracture: physiologic aspects of formation</atitle><jtitle>Clinical orthopaedics and related research</jtitle><addtitle>Clin Orthop Relat Res</addtitle><date>1988-08-01</date><risdate>1988</risdate><volume>233</volume><issue>233</issue><spage>7</spage><epage>18</epage><pages>7-18</pages><issn>0009-921X</issn><eissn>1528-1132</eissn><coden>CORTBR</coden><abstract>Disruption of the upper motor neuron inhibitory pathways by stroke, brain trauma, or spinal cord injury leads to muscle spasticity. Spasticity is characterized by increased muscle tone, hyperactive reflexes, and possible clonus or rigidity. The increased muscle tone may result in loss of joint motion, leading to contractures. Treatment of established contractures is difficult. Prevention of contractures by joint mobilization is emphasized as a goal in the management of patients with spasticity.</abstract><cop>Heidelberg</cop><pub>Springer</pub><pmid>3042237</pmid><doi>10.1097/00003086-198808000-00003</doi><tpages>12</tpages></addata></record> |
fulltext | fulltext |
identifier | ISSN: 0009-921X |
ispartof | Clinical orthopaedics and related research, 1988-08, Vol.233 (233), p.7-18 |
issn | 0009-921X 1528-1132 |
language | eng |
recordid | cdi_proquest_miscellaneous_78355335 |
source | MEDLINE; Journals@Ovid Complete |
subjects | Animals Biological and medical sciences Contracture - physiopathology Contracture - prevention & control Contracture - therapy Humans Medical sciences Motor Neurons - physiology Muscle Spasticity - physiopathology Muscle Spasticity - prevention & control Muscle Spasticity - therapy Muscles - innervation Nervous system (semeiology, syndromes) Nervous system as a whole Neural Pathways - anatomy & histology Neural Pathways - physiology Neurology Neurons, Efferent - physiology |
title | Spasticity and contracture: physiologic aspects of formation |
url | https://sfx.bib-bvb.de/sfx_tum?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2024-12-13T08%3A01%3A30IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-proquest_cross&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=Spasticity%20and%20contracture:%20physiologic%20aspects%20of%20formation&rft.jtitle=Clinical%20orthopaedics%20and%20related%20research&rft.au=BOTTE,%20M.%20J&rft.date=1988-08-01&rft.volume=233&rft.issue=233&rft.spage=7&rft.epage=18&rft.pages=7-18&rft.issn=0009-921X&rft.eissn=1528-1132&rft.coden=CORTBR&rft_id=info:doi/10.1097/00003086-198808000-00003&rft_dat=%3Cproquest_cross%3E78355335%3C/proquest_cross%3E%3Curl%3E%3C/url%3E&disable_directlink=true&sfx.directlink=off&sfx.report_link=0&rft_id=info:oai/&rft_pqid=78355335&rft_id=info:pmid/3042237&rfr_iscdi=true |