Transcriptional Induction of Rat Liver Apolipoprotein A‐I Gene Expression by Glucocorticoids Requires the Glucocorticoid Receptor and a Labile Cell‐Specific Protein

Treatment with glucocorticoids increases the concentration of plasma high‐density lipoprotein (HDL), which is inversely correlated to the development of atherosclerosis. Previously, we demonstrated that repeated administration of glucocorticoids increases apolipoprotein (apo) A‐I gene expression and...

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Veröffentlicht in:	European journal of biochemistry 1996-07, Vol.239 (2), p.451-459
Hauptverfasser:	Saladin, Régis, Vu‐Dac, Ngoc, Fruchart, Jean‐Charles, Auwerx, Johan, Staels, Bart
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals apolipoprotein Apolipoprotein A-I - biosynthesis Apolipoprotein A-II - biosynthesis Base Sequence Carcinoma, Hepatocellular Cell Line Cell Nucleus - metabolism Cells, Cultured Cercopithecus aethiops Chloramphenicol O-Acetyltransferase - biosynthesis Cloning, Molecular corticosteroid hormone Dactinomycin - pharmacology Dexamethasone - pharmacology DNA Primers gene expression Glucocorticoids - pharmacology Kinetics liver Liver - drug effects Liver - metabolism Male Mifepristone - pharmacology Molecular Sequence Data Polymerase Chain Reaction Rats Rats, Sprague-Dawley Receptors, Glucocorticoid - drug effects Receptors, Glucocorticoid - physiology Recombinant Proteins - biosynthesis RNA, Messenger - biosynthesis Transcription, Genetic - drug effects Transfection
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