Temporal regulation by adrenergic receptor stimulation of macrophage ( MΦ)-derived tumor necrosis factor (TNF) production post-LPS challenge

Macrophage (MΦ) responsiveness can be regulated by various mediators, including those which emanate from, and mimic, the sympathetic nervous system. Whereas β-adrenergic agonists suppress, α 2-adrenergic agonists augment lipopolysaccharide (LPS)-stimulated tumor necrosis factor (TNF) production and...

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Veröffentlicht in:	Journal of neuroimmunology 1996-04, Vol.65 (2), p.107-117
Hauptverfasser:	Ignatowski, Tracey A., Gallant, Samuel, Spengler, Robert N.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adrenergic Adrenergic alpha-Agonists - pharmacology Adrenergic beta-Agonists - pharmacology Animals Base Sequence Bucladesine - pharmacology Cells, Cultured Female Isoproterenol - pharmacology Lipopolysaccharide Lipopolysaccharides - pharmacology Macrophage Macrophages - metabolism Mice Mice, Inbred CBA Molecular Sequence Data Oligonucleotide Probes - genetics Osmolar Concentration Receptors, Adrenergic - physiology RNA, Messenger - metabolism Sympathetic nervous system Time Factors Tumor necrosis factor Tumor Necrosis Factor-alpha - biosynthesis Tumor Necrosis Factor-alpha - genetics
Online-Zugang:	Volltext
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