Stress‐induced hemodynamic and hemostatic changes in patients with systemic hypertension: Effect of verapamil

Stress‐induced hemodynamic and hemostatic responses may acutely trigger atherosclerotic plaque disruption and thrombosis leading to myocardial infarction. This study was designed to evaluate the responses to three stressors and to determine if once‐daily sustained release verapamil (Verelan®) modifi...

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Veröffentlicht in:	Clinical cardiology (Mahwah, N.J.) N.J.), 1996-03, Vol.19 (3), p.205-211
Hauptverfasser:	Gebara, Otavio C. E., Jimenez, Alfredo H., Mckenna, Carol, Mittleman, Murray A., Xu, Ping, Lipinska, Izabella, Muller, James E., Tofler, Geoffrey H.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenosine Diphosphate - pharmacology Adult Antihypertensive agents Biological and medical sciences Blood Pressure - drug effects Blood Pressure - physiology Calcium Channel Blockers - administration & dosage Calcium Channel Blockers - therapeutic use calcium‐channel blocker Cardiovascular system Collagen - pharmacology Cross-Over Studies Delayed-Action Preparations Double-Blind Method Epinephrine - pharmacology Female Heart Rate - drug effects Heart Rate - physiology Humans hypertension Hypertension - drug therapy Hypertension - physiopathology Male Medical sciences Middle Aged Pharmacology. Drug treatments Placebos Platelet Activation - drug effects Platelet Activation - physiology Platelet Aggregation - drug effects Platelet Aggregation - physiology platelets Posture Stress, Physiological - physiopathology Stress, Psychological - physiopathology Vasodilator Agents - administration & dosage Vasodilator Agents - therapeutic use Verapamil - administration & dosage Verapamil - therapeutic use
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