Altered 11β-hydroxylase activity in glucocorticoid-suppressible hyperaldosteronism

Corticosteroid 11 beta-hydroxylation is catalyzed by 11 beta-hydroxylase and aldosterone synthase. Using plasma steroid ratios, the level of this process in patients with glucocorticoid-suppressible hyperaldosteronism (GSH) was compared with that in unaffected control subjects and patients with Conn...

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Veröffentlicht in:	The journal of clinical endocrinology and metabolism 1996-06, Vol.81 (6), p.2298-2302
Hauptverfasser:	JAMIESON, A, INGRAM, M. C, INGLIS, G. C, DAVIES, E, FRASER, R, CONNELL, J. M. C
Format:	Artikel
Sprache:	eng
Schlagworte:	Adrenal Cortex Neoplasms - complications Adrenal Cortex Neoplasms - secretion Adrenals. Adrenal axis. Renin-angiotensin system (diseases) Aldosterone - secretion Biological and medical sciences Corticosterone - blood Cortodoxone - blood Desoxycorticosterone - blood Endocrinopathies Glucocorticoids - therapeutic use Humans Hydrocortisone - blood Hyperaldosteronism - blood Hyperaldosteronism - drug therapy Hyperaldosteronism - genetics Medical sciences Non tumoral diseases. Target tissue resistance. Benign neoplasms Steroid 11-beta-Hydroxylase - blood
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