The inward rectification mechanism of the HERG cardiac potassium channel

A HUMAN genetic defect associated with 'long Q–T syndrome', an abnormality of cardiac rhythm involving the repolarization of the action potential, was recently found to lie in the HERG gene, which codes for a potassium channel 1 . The HERG K + channel is unusual in that it seems to have th...

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Veröffentlicht in:	Nature (London) 1996-02, Vol.379 (6568), p.833-836
Hauptverfasser:	Smith, Paula L., Baukrowitz, Thomas, Yellen, Gary
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Arrhythmias, Cardiac - metabolism Biochemistry Biological and medical sciences Cardiac arrhythmia Cation Transport Proteins Cell Line Cell membranes. Ionic channels. Membrane pores Cell structures and functions DNA-Binding Proteins Drosophila Drosophila Proteins ERG1 Potassium Channel Ether-A-Go-Go Potassium Channels Fundamental and applied biological sciences. Psychology Humanities and Social Sciences Humans Inactivation Ion Channel Gating Ions letter Long QT Syndrome - metabolism Magnesium - pharmacology Mammals Membrane Potentials Molecular and cellular biology multidisciplinary Mutation Myocardium - metabolism Potassium Potassium Channel Blockers Potassium Channels - genetics Potassium Channels - metabolism Potassium Channels, Voltage-Gated Recombinant Proteins Science Science (multidisciplinary) Shaker Superfamily of Potassium Channels Tetraethylammonium Tetraethylammonium Compounds - pharmacology Trans-Activators Transcriptional Regulator ERG
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